Michael D. Henry

Dystroglycan: an extracellular matrix receptor linked to the cytoskeleton.

Dystroglycan inside and out.

Dystroglycan is selectively associated with inhibitory GABAergic synapses but is dispensable for their differentiation.

Deletion of brain dystroglycan recapitulates aspects of congenital muscular dystrophy.

Unique role of dystroglycan in peripheral nerve myelination, nodal structure, and sodium channel stabilization.

Spiculated periosteal response induced by intraosseous injection of 22Rv1 prostate cancer cells resembles subset of bone metastases in prostate cancer patients.

Disruption of perlecan binding and matrix assembly by post-translational or genetic disruption of dystroglycan function.

Assessing tumor growth and distribution in a model of prostate cancer metastasis using bioluminescence imaging.

Dystroglycan loss disrupts polarity and beta-casein induction in mammary epithelial cells by perturbing laminin anchoring.

Chemotherapeutic agents up-regulate the cytomegalovirus promoter: implications for bioluminescence imaging of tumor response to therapy.

Assessing siRNA pharmacodynamics in a luciferase-expressing mouse.

ZEB1 enhances transendothelial migration and represses the epithelial phenotype of prostate cancer cells.

Endothelin-1 inhibits prostate cancer growth in vivo through vasoconstriction of tumor-feeding arterioles.

Loss of alpha-dystroglycan laminin binding in epithelium-derived cancers is caused by silencing of LARGE.

Dystroglycan is not required for maintenance of the luminal epithelial basement membrane or cell polarity in the mouse prostate.

Bone-specific growth inhibition of prostate cancer metastasis by atrasentan.

ZEB1 coordinately regulates laminin-332 and {beta}4 integrin expression altering the invasive phenotype of prostate cancer cells.

The ARF tumor suppressor inhibits tumor cell colonization independent of p53 in a novel mouse model of pancreatic ductal adenocarcinoma metastasis.

An inducible model of abacterial prostatitis induces antigen specific inflammatory and proliferative changes in the murine prostate.

Epithelial-to-mesenchymal transition in prostate cancer: paradigm or puzzle?

Slow disease progression in a C57BL/6 pten-deficient mouse model of prostate cancer.

Eradication of metastatic renal cell carcinoma after adenovirus-encoded TNF-related apoptosis-inducing ligand (TRAIL)/CpG immunotherapy.

Loss of LARGE2 disrupts functional glycosylation of α-dystroglycan in prostate cancer.

Integrin α3β1 regulates tumor cell responses to stromal cells and can function to suppress prostate cancer metastatic colonization.

Resistance to fluid shear stress is a conserved biophysical property of malignant cells.

Chronic chlorpyrifos exposure does not promote prostate cancer in prostate specific PTEN mutant mice.

Integrin α3β1 can function to promote spontaneous metastasis and lung colonization of invasive breast carcinoma.

Prolyl-4-hydroxylase 3 (PHD3) expression is downregulated during epithelial-to-mesenchymal transition.

Transcriptome-wide landscape of pre-mRNA alternative splicing associated with metastatic colonization.

Impact of prostate inflammation on lesion development in the POET3(+)Pten(+/-) mouse model of prostate carcinogenesis.

The glycosyltransferase LARGE2 is repressed by Snail and ZEB1 in prostate cancer.

rMATS: robust and flexible detection of differential alternative splicing from replicate RNA-Seq data.

Loss of SOD3 (EcSOD) Expression Promotes an Aggressive Phenotype in Human Pancreatic Ductal Adenocarcinoma.

Alterations in cancer cell mechanical properties after fluid shear stress exposure: a micropipette aspiration study.

Targeting geranylgeranylation reduces adrenal gland tumor burden in a murine model of prostate cancer metastasis.

Downregulation of dystroglycan glycosyltransferases LARGE2 and ISPD associate with increased mortality in clear cell renal cell carcinoma.

Individualization of Adjuvant Therapy After Radical Prostatectomy for Clinically Localized Prostate Cancer: Current Status and Future Directions.

CD151 promotes α3β1 integrin-dependent organization of carcinoma cell junctions and restrains collective cell invasion.

α3β1 Integrin Suppresses Prostate Cancer Metastasis via Regulation of the Hippo Pathway.

Inhibiting G protein βγ signaling blocks prostate cancer progression and enhances the efficacy of paclitaxel.

Natural Products Discovered in a High-Throughput Screen Identified as Inhibitors of RGS17 and as Cytostatic and Cytotoxic Agents for Lung and Prostate Cancer Cell Lines.

A Trp53fl/flPtenfl/fl mouse model of undifferentiated pleomorphic sarcoma mediated by adeno-Cre injection and in vivo bioluminescence imaging.

Prostate tumor cell exosomes containing hyaluronidase Hyal1 stimulate prostate stromal cell motility by engagement of FAK-mediated integrin signaling.

Biomechanics of the Circulating Tumor Cell Microenvironment.

High content screening identifies monensin as an EMT-selective cytotoxic compound.

A model for the detection of pancreatic ductal adenocarcinoma circulating tumor cells.

Alpha-dystroglycan staining pattern and mortality in patients undergoing radical prostatectomy with lymph node positive prostate cancer.

Cancer Cells Resist Mechanical Destruction in Circulation via RhoA/Actomyosin-Dependent Mechano-Adaptation.

Functional Genomic Screening Independently Identifies CUL3 as a Mediator of Vemurafenib Resistance via Src-Rac1 Signaling Axis.

Survival of the resilient: Mechano-adaptation of circulating tumor cells to fluid shear stress.

Locally invasive, castrate-resistant prostate cancer in a Pten/Trp53 double knockout mouse model of prostate cancer monitored with non-invasive bioluminescent imaging.

Pharmacological ascorbate inhibits pancreatic cancer metastases via a peroxide-mediated mechanism.

Melanoma Brain Metastases in the Era of Targeted Therapy and Checkpoint Inhibitor Therapy.