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Keren K. Griffiths

Department of Anesthesiology

Columbia University Medical Center

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PUBLICATIONS

Evidence of Mitochondrial Dysfunction in Autism: Biochemical Links, Genetic-Based Associations, and Non-Energy-Related Mechanisms.

Oxidative medicine and cellular longevity , 2017 | Pubmed ID: 28630658

Inefficient thermogenic mitochondrial respiration due to futile proton leak in a mouse model of fragile X syndrome.

FASEB journal : official publication of the Federation of American Societies for Experimental Biology 06, 2020 | Pubmed ID: 32307754

The newborn Fmr1 knockout mouse: a novel model of excess ubiquinone and closed mitochondrial permeability transition pore in the developing heart.

Pediatric research 02, 2021 | Pubmed ID: 32674111

Insights image for "The newborn Fmr1 knockout mouse: a novel model of excess ubiquinone and closed mitochondrial permeability transition pore in the developing heart".

Pediatric research 02, 2021 | Pubmed ID: 32919395

Propofol toxicity in the developing mouse heart mitochondria.

Pediatric research Feb, 2022 | Pubmed ID: 35173299

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Columbia University Medical Center

JOVE ARTICLES

Assessment of Open Probability of the Mitochondrial Permeability Transition Pore in the Setting of Coenzyme Q Excess

Keren K. Griffiths¹,

Aili Wang¹,

Richard J. Levy¹

¹Department of Anesthesiology, Columbia University Medical Center

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Keren K. Griffiths

.css-o250i3{font-size:18px;font-family:Open Sans,sans-serif;line-height:21.6px;}Department of Anesthesiology

Columbia University Medical Center

Assessment of Open Probability of the Mitochondrial Permeability Transition Pore in the Setting of Coenzyme Q Excess

Department of Anesthesiology