Florent Elefteriou

Leptin regulates bone formation via the sympathetic nervous system.

Common endocrine control of body weight, reproduction, and bone mass.

Reversible skeletal abnormalities in gamma-glutamyl transpeptidase-deficient mice.

[Control of body mass by leptin occurs through the sympathetic nervous system].

Monosodium glutamate-sensitive hypothalamic neurons contribute to the control of bone mass.

Essential role of STAT3 in body weight and glucose homeostasis.

Enhanced bone formation in lipodystrophic PPARgamma(hyp/hyp) mice relocates haematopoiesis to the spleen.

Leptin regulation of bone resorption by the sympathetic nervous system and CART.

Boning up on ephrin signaling.

ATF4 mediation of NF1 functions in osteoblast reveals a nutritional basis for congenital skeletal dysplasiae.

Regulation of bone remodeling by the central and peripheral nervous system.

Cocaine and amphetamine-regulated transcript may regulate bone remodeling as a circulating molecule.

Vimentin inhibits ATF4-mediated osteocalcin transcription and osteoblast differentiation.

Skeletal abnormalities in neurofibromatosis type 1: approaches to therapeutic options.

Atf4 regulates chondrocyte proliferation and differentiation during endochondral ossification by activating Ihh transcription.

Local low-dose lovastatin delivery improves the bone-healing defect caused by Nf1 loss of function in osteoblasts.

β2-Adrenergic receptor signaling in osteoblasts contributes to the catabolic effect of glucocorticoids on bone.

Mice lacking Nf1 in osteochondroprogenitor cells display skeletal dysplasia similar to patients with neurofibromatosis type I.

Genetic mouse models for bone studies--strengths and limitations.

Chondrocytic Atf4 regulates osteoblast differentiation and function via Ihh.

Stimulation of host bone marrow stromal cells by sympathetic nerves promotes breast cancer bone metastasis in mice.