Ipek Yalcin

Dual effects of nitric oxide in the mouse forced swimming test: possible contribution of nitric oxide-mediated serotonin release and potassium channel modulation.

Involvement of potassium channels and nitric oxide in tramadol antinociception.

Effects of desipramine and tramadol in a chronic mild stress model in mice are altered by yohimbine but not by pindolol.

Rho-kinase inhibitor, Y-27632, has an antinociceptive effect in mice.

Delta-opioid receptors are critical for tricyclic antidepressant treatment of neuropathic allodynia.

Antidepressant-like effect of tramadol in the unpredictable chronic mild stress procedure: possible involvement of the noradrenergic system.

Chronic, but not acute, tricyclic antidepressant treatment alleviates neuropathic allodynia after sciatic nerve cuffing in mice.

Mouse strain differences in the unpredictable chronic mild stress: a four-antidepressant survey.

Effects of 5,7-dihydroxytryptamine lesion of the dorsal raphe nucleus on the antidepressant-like action of tramadol in the unpredictable chronic mild stress in mice.

Beta2-adrenoceptors are essential for desipramine, venlafaxine or reboxetine action in neuropathic pain.

beta(2)-adrenoceptors are critical for antidepressant treatment of neuropathic pain.

Differentiating thermal allodynia and hyperalgesia using dynamic hot and cold plate in rodents.

From antidepressant drugs to beta-mimetics: preclinical insights on potential new treatments for neuropathic pain.

Implication of beta3-adrenoceptors in the antidepressant-like effects of amibegron using Adrb3 knockout mice in the chronic mild stress.

Chronic treatment with agonists of beta(2)-adrenergic receptors in neuropathic pain.

Beta2-adrenoceptor agonists alleviate neuropathic allodynia in mice after chronic treatment.

Mu-opioid receptors are not necessary for nortriptyline treatment of neuropathic allodynia.

Is there a place for beta-mimetics in clinical management of neuropathic pain? Salbutamol therapy in six cases.

Cardiovascular effects of chronic treatment with a β2-adrenoceptor agonist relieving neuropathic pain in mice.

Nociceptive thresholds are controlled through spinal β2-subunit-containing nicotinic acetylcholine receptors.

A time-dependent history of mood disorders in a murine model of neuropathic pain.

BDNF parabrachio-amygdaloid pathway in morphine-induced analgesia.

Antidepressants suppress neuropathic pain by a peripheral β2-adrenoceptor mediated anti-TNFα mechanism.