Jennifer L. Strande

SCH 79797, a selective PAR1 antagonist, limits myocardial ischemia/reperfusion injury in rat hearts.

Human thrombopoietin reduces myocardial infarct size, apoptosis, and stunning following ischaemia/reperfusion in rats.

Inhibiting protease-activated receptor 4 limits myocardial ischemia/reperfusion injury in rat hearts by unmasking adenosine signaling.

Gadolinium limits myocardial infarction in the rat: dose-response, temporal relations and mechanisms.

Letter by Strande regarding article "Protease-activated receptor-1 contributes to cardiac remodeling and hypertrophy".

Thrombin increases inflammatory cytokine and angiogenic growth factor secretion in human adipose cells in vitro.

Parstatin: a cryptic peptide involved in cardioprotection after ischaemia and reperfusion injury.

10 Gy total body irradiation increases risk of coronary sclerosis, degeneration of heart structure and function in a rat model.

Gadolinium decreases inflammation related to myocardial ischemia and reperfusion injury.

Parstatin(1-26): the putative signal peptide of protease-activated receptor 1 confers potent protection from myocardial ischemia-reperfusion injury.

Nerve growth factor reduces myocardial ischemia/reperfusion injury in rat hearts.

Protease-activated receptor 1 inhibition by SCH79797 attenuates left ventricular remodeling and profibrotic activities of cardiac fibroblasts.

Dystrophin-deficient cardiomyocytes derived from human urine: new biologic reagents for drug discovery.

Whole-thorax irradiation induces hypoxic respiratory failure, pleural effusions and cardiac remodeling.

Haploinsufficiency MYBPC3 mutations: Another stress induced cardiomyopathy? Let's take a look!