Yanhua Hu

Loss of p53 accelerates neointimal lesions of vein bypass grafts in mice.

Mechanical stress-induced DNA damage and rac-p38MAPK signal pathways mediate p53-dependent apoptosis in vascular smooth muscle cells.

Smooth muscle cells in transplant atherosclerotic lesions are originated from recipients, but not bone marrow progenitor cells.

Both donor and recipient origins of smooth muscle cells in vein graft atherosclerotic lesions.

Experimental study of plasmid TGF-beta 1 DNA gene transfer with lipofectamine into rabbit corneal epithelial cells in vitro.

Development and clinical application of an quantitative head-band formed ocular compressor.

The expression of the plasmid DNA encoding TGF-beta 1 in endothelium after injection into the anterior chamber.

Activation of heat shock transcription factor 1 in atherosclerosis.

Mechanical stress-activated PKCdelta regulates smooth muscle cell migration.

Circulating progenitor cells regenerate endothelium of vein graft atherosclerosis, which is diminished in ApoE-deficient mice.

Endothelial replacement and angiogenesis in arteriosclerotic lesions of allografts are contributed by circulating progenitor cells.

Thrombosis and neointima formation in vein grafts are inhibited by locally applied aspirin through endothelial protection.

Abundant progenitor cells in the adventitia contribute to atherosclerosis of vein grafts in ApoE-deficient mice.

Automated interpretation of subcellular patterns from immunofluorescence microscopy.

Ischemic preconditioning exaggerates cardiac damage in PKC-delta null mice.

Expression of VEGF-C in rat cornea after alkali injury.

Biomechanical stress induces IL-6 expression in smooth muscle cells via Ras/Rac1-p38 MAPK-NF-kappaB signaling pathways.

Adventitial progenitor cells contribute to arteriosclerosis.

Ipsilateral lymphadenectomy to inhibit corneal allograft rejection in rats.

Proteomic and metabolomic analyses of atherosclerotic vessels from apolipoprotein E-deficient mice reveal alterations in inflammation, oxidative stress, and energy metabolism.

Gene transfer of a broad spectrum CC-chemokine inhibitor reduces vein graft atherosclerosis in apolipoprotein E-knockout mice.

New mouse model of vein bypass graft atherosclerosis.

Accelerated arteriosclerosis of vein grafts in inducible NO synthase(-/-) mice is related to decreased endothelial progenitor cell repair.

Central corneal thickness in adult Chinese.

TLR4 expression in mouse embryonic stem cells and in stem cell-derived vascular cells is regulated by epigenetic modifications.

Lymphangiogenesis occurring in transplanted corneas.

Sca-1+ progenitors derived from embryonic stem cells differentiate into endothelial cells capable of vascular repair after arterial injury.

Stem cell-derived Sca-1+ progenitors differentiate into smooth muscle cells, which is mediated by collagen IV-integrin alpha1/beta1/alphav and PDGF receptor pathways.

Effects of CTLA4-Ig gene-modified dendritic cells on the corneal allografts.

HDAC3 is crucial in shear- and VEGF-induced stem cell differentiation toward endothelial cells.

Increased in-stent stenosis in ApoE knockout mice: insights from a novel mouse model of balloon angioplasty and stenting.

Effects of shRNA targeting survivin on apoptosis of human retinoblastoma cell line Hxo-rb44 in vitro.

Resection and cryotherapy combined with amniotic membrane transplantation for the treatment of vernal keratoconjunctivitis with giant papillae.

Lacking cytokine production in ES cells and ES-cell-derived vascular cells stimulated by TNF-alpha is rescued by HDAC inhibitor trichostatin A.

Endothelial progenitor cells, cardiovascular risk factors, cytokine levels and atherosclerosis--results from a large population-based study.

Vessel graft atherosclerosis in murine models.

Rapid endothelial turnover in atherosclerosis-prone areas coincides with stem cell repair in apolipoprotein E-deficient mice.

Proteomic and metabolomic analysis of smooth muscle cells derived from the arterial media and adventitial progenitors of apolipoprotein E-deficient mice.

A comparative assessment of the efficacy of carbomer gel and carboxymethyl cellulose containing artificial tears in dry eyes.

Splicing of HDAC7 modulates the SRF-myocardin complex during stem-cell differentiation towards smooth muscle cells.

Sustained activation of XBP1 splicing leads to endothelial apoptosis and atherosclerosis development in response to disturbed flow.

A role of matrix metalloproteinase-8 in atherosclerosis.

Histone deacetylase 3 is critical in endothelial survival and atherosclerosis development in response to disturbed flow.

Crucial role of nrf3 in smooth muscle cell differentiation from stem cells.

Histone deacetylase 7 controls endothelial cell growth through modulation of beta-catenin.

Increased sensitivity of HIV variants selected by attachment inhibitors to broadly neutralizing antibodies.

Automated analysis of protein subcellular location in time series images.

Different effects of angiotensin II and angiotensin-(1-7) on vascular smooth muscle cell proliferation and migration.

Phosphorylation of threonine 154 in p40phox is an important physiological signal for activation of the neutrophil NADPH oxidase.

Protein kinase C{delta} deficiency accelerates neointimal lesions of mouse injured artery involving delayed reendothelialization and vasohibin-1 accumulation.

Topical HDL administration reduces vein graft atherosclerosis in apo E deficient mice.

Adventitial biology: differentiation and function.

Splicing of histone deacetylase 7 modulates smooth muscle cell proliferation and neointima formation through nuclear β-catenin translocation.

Galectin-9 protein expression in endothelial cells is positively regulated by histone deacetylase 3.

Nrf3-Pla2g7 Interaction Plays an Essential Role in Smooth Muscle Cell Differentiation From Stem Cells.

A templated method to Bi2WO6 hollow microspheres and their conversion to double-shell Bi2O3/Bi2WO6 hollow microspheres with improved photocatalytic performance.

Contribution of stem cells to neointimal formation of decellularized vessel grafts in a novel mouse model.

Direct reprogramming of fibroblasts into endothelial cells capable of angiogenesis and reendothelialization in tissue-engineered vessels.

XBP1 mRNA splicing triggers an autophagic response in endothelial cells through BECLIN-1 transcriptional activation.

Smooth muscle cells differentiated from reprogrammed embryonic lung fibroblasts through DKK3 signaling are potent for tissue engineering of vascular grafts.

Vascular endothelial cell growth-activated XBP1 splicing in endothelial cells is crucial for angiogenesis.

Allogeneic bone marrow-derived mesenchymal stem cells attenuate hepatic ischemia-reperfusion injury by suppressing oxidative stress and inhibiting apoptosis in rats.

Adventitial stem cells in vein grafts display multilineage potential that contributes to neointimal formation.

Dysfunction of endothelial progenitor cells from smokers and chronic obstructive pulmonary disease patients due to increased DNA damage and senescence.

Tetrahydrobiopterin determines vascular remodeling through enhanced endothelial cell survival and regeneration.

Histone deacetylase 3 unconventional splicing mediates endothelial-to-mesenchymal transition through transforming growth factor β2.

Matrix metalloproteinase-8 promotes vascular smooth muscle cell proliferation and neointima formation.

Endothelial lineage differentiation from induced pluripotent stem cells is regulated by microRNA-21 and transforming growth factor β2 (TGF-β2) pathways.

Macrophages control vascular stem/progenitor cell plasticity through tumor necrosis factor-α-mediated nuclear factor-κB activation.

Stem cells accumulate on a decellularized arterial xenograft in vivo.

Bidirectional cross-regulation between the endothelial nitric oxide synthase and β-catenin signalling pathways.

Unspliced X-box-binding protein 1 (XBP1) protects endothelial cells from oxidative stress through interaction with histone deacetylase 3.

MicroRNA-199b modulates vascular cell fate during iPS cell differentiation by targeting the notch ligand jagged1 and enhancing VEGF signaling.