Fatah Kashanchi

Human T-lymphotropic virus type 1 transcription and chromatin-remodeling complexes.

Inhibition of human immunodeficiency virus type-1 by cdk inhibitors.

Absence of DICER in monocytes and its regulation by HIV-1.

Transcription through the HIV-1 nucleosomes: effects of the PBAF complex in Tat activated transcription.

The identification of unique serum proteins of HIV-1 latently infected long-term non-progressor patients.

Methylation of the tumor suppressor protein, BRCA1, influences its transcriptional cofactor function.

microRNA machinery is an integral component of drug-induced transcription inhibition in HIV-1 infection.

Direct detection of diverse metabolic changes in virally transformed and tax-expressing cells by mass spectrometry.

Iron chelators of the di-2-pyridylketone thiosemicarbazone and 2-benzoylpyridine thiosemicarbazone series inhibit HIV-1 transcription: identification of novel cellular targets--iron, cyclin-dependent kinase (CDK) 2, and CDK9.

Erratum to: microRNA machinery is an integral component of drug-induced transcription inhibition in HIV-1 infection.

Reverse-phase phosphoproteome analysis of signaling pathways induced by Rift valley fever virus in human small airway epithelial cells.

Hepcidin induces HIV-1 transcription inhibited by ferroportin.

Analysis of the roles of HIV-derived microRNAs.

Developments in antivirals against influenza, smallpox and hemorrhagic fever viruses.

Secreted Bacillus anthracis proteases target the host fibrinolytic system.

Bacillus anthracis interacts with plasmin(ogen) to evade C3b-dependent innate immunity.

Inhibition of Tat-mediated HIV-1 replication and neurotoxicity by novel GSK3-beta inhibitors.

Alteration in superoxide dismutase 1 causes oxidative stress and p38 MAPK activation following RVFV infection.

Targeting the inflammasome and adenosine type-3 receptors improves outcome of antibiotic therapy in murine anthrax.

Varying modulation of HIV-1 LTR activity by Baf complexes.

Role of β-catenin and TCF/LEF family members in transcriptional activity of HIV in astrocytes.

Humanized mouse models of HIV-1 latency.

Induction of DNA damage signaling upon Rift Valley fever virus infection results in cell cycle arrest and increased viral replication.

Liver X receptor agonist inhibits HIV-1 replication and prevents HIV-induced reduction of plasma HDL in humanized mouse model of HIV infection.

New and novel intrinsic host repressive factors against HIV-1: PAF1 complex, HERC5 and others.

Bacillus anthracis protease InhA regulates BslA-mediated adhesion in human endothelial cells.

Modulation of GSK-3β activity in Venezuelan equine encephalitis virus infection.

p53 Activation following Rift Valley fever virus infection contributes to cell death and viral production.

Identification of novel T cell factor 4 (TCF-4) binding sites on the HIV long terminal repeat which associate with TCF-4, β-catenin, and SMAR1 to repress HIV transcription.

Use of ATP analogs to inhibit HIV-1 transcription.

Localization and sub-cellular shuttling of HTLV-1 tax with the miRNA machinery.

Enduring cortical alterations after a single in-vivo treatment of HIV-1 Tat.

Curcumin inhibits Rift Valley fever virus replication in human cells.

In vivo murine and in vitro M-like cell models of gastrointestinal anthrax.

CDK2 regulates HIV-1 transcription by phosphorylation of CDK9 on serine 90.

Bacillus anthracis-derived nitric oxide induces protein S-nitrosylation contributing to macrophage death.

HTLV tax: a fascinating multifunctional co-regulator of viral and cellular pathways.

Effect of Mimetic CDK9 Inhibitors on HIV-1-Activated Transcription.

Complex role of microRNAs in HTLV-1 infections.

Retrovirus infected cells contain viral microRNAs.

Exosomes derived from HIV-1-infected cells contain trans-activation response element RNA.

The use of NanoTrap particles as a sample enrichment method to enhance the detection of Rift Valley Fever Virus.

Novel neuroprotective GSK-3β inhibitor restricts Tat-mediated HIV-1 replication.

Reactive oxygen species activate NFκB (p65) and p53 and induce apoptosis in RVFV infected liver cells.

The carrying pigeons of the cell: exosomes and their role in infectious diseases caused by human pathogens.

Repeated cocaine treatment enhances HIV-1 Tat-induced cortical excitability via over-activation of L-type calcium channels.

Exosomes and their role in CNS viral infections.

The role of IKKβ in Venezuelan equine encephalitis virus infection.

The use of Nanotrap particles technology in capturing HIV-1 virions and viral proteins from infected cells.

Transcriptional Gene Silencing (TGS) via the RNAi Machinery in HIV-1 Infections.

Human T-lymphotropic virus type 1-infected cells secrete exosomes that contain Tax protein.

Phenyl-1-Pyridin-2yl-ethanone-based iron chelators increase IκB-α expression, modulate CDK2 and CDK9 activities, and inhibit HIV-1 transcription.

Break CDK2/Cyclin E1 interface allosterically with small peptides.

Discovery and characterization of auxiliary proteins encoded by type 3 simian T-cell lymphotropic viruses.

Role of Bruton's tyrosine kinase inhibitors in HIV-1-infected cells.

Cocaine promotes both initiation and elongation phase of HIV-1 transcription by activating NF-κB and MSK1 and inducing selective epigenetic modifications at HIV-1 LTR.

Reactivation of latent HIV-1 provirus via targeting protein phosphatase-1.

Therapeutic doses of irradiation activate viral transcription and induce apoptosis in HIV-1 infected cells.

Extracellular vesicles from infected cells: potential for direct pathogenesis.

Exosomes from HIV-1-infected Cells Stimulate Production of Pro-inflammatory Cytokines through Trans-activating Response (TAR) RNA.

Improvements and Limitations of Humanized Mouse Models for HIV Research: NIH/NIAID "Meet the Experts" 2015 Workshop Summary.

HIV-1 Nef blocks autophagy in human astrocytes.

Potential of Radiation-Induced Cellular Stress for Reactivation of Latent HIV-1 and Killing of Infected Cells.

Presence of Viral RNA and Proteins in Exosomes from Cellular Clones Resistant to Rift Valley Fever Virus Infection.

Zika Virus: A New Animal Model for an Arbovirus.

Exosomes in Viral Disease.

Biodegradable Nanoparticles for Delivery of Therapeutics in CNS Infection.

HIV Vpr controls CNS metabolism.

Ebola VP40 in Exosomes Can Cause Immune Cell Dysfunction.

The Role of Exosomal VP40 in Ebola Virus Disease.

The Human Immunodeficiency Virus 1 ASP RNA promotes viral latency by recruiting the Polycomb Repressor Complex 2 and promoting nucleosome assembly.

Isolation of Exosomes from HTLV-Infected Cells.

Presence of Viral microRNA in Extracellular Environments.

Intranasal drug delivery of small interfering RNA targeting Beclin1 encapsulated with polyethylenimine (PEI) in mouse brain to achieve HIV attenuation.

Exosomes from uninfected cells activate transcription of latent HIV-1.

HIV-1 Transcription Inhibitors Increase the Synthesis of Viral Non-Coding RNA that Contribute to Latency.

Interplay between Autophagy, Exosomes and HIV-1 Associated Neurological Disorders: New Insights for Diagnosis and Therapeutic Applications.

Importance of Autophagy in Mediating Human Immunodeficiency Virus (HIV) and Morphine-Induced Metabolic Dysfunction and Inflammation in Human Astrocytes.

Proceedings of the ISEV symposium on "HIV, NeuroAIDS, drug abuse & EVs".

Inhibition of HIV-1 infection in humanized mice and metabolic stability of protein phosphatase-1-targeting small molecule 1E7-03.

Proceedings of the 2017 ISEV symposium on "HIV, NeuroHIV, drug abuse, & EVs".

Monocyte-derived exosomes upon exposure to cigarette smoke condensate alter their characteristics and show protective effect against cytotoxicity and HIV-1 replication.

Author Correction: Intranasal drug delivery of small interfering RNA targeting Beclin1 encapsulated with polyethylenimine (PEI) in mouse brain to achieve HIV attenuation.

Tat controls transcriptional persistence of unintegrated HIV genome in primary human macrophages.

Advances, challenges, and opportunities in extracellular RNA biology: insights from the NIH exRNA Strategic Workshop.

Role of Exosomes in Human Retroviral Mediated Disorders.

Corrigendum: Ebola VP40 in Exosomes Can Cause Immune Cell Dysfunction.

Antiretroviral Drugs Alter the Content of Extracellular Vesicles from HIV-1-Infected Cells.

Viral antigens detectable in CSF exosomes from patients with retrovirus associated neurologic disease: functional role of exosomes.

Ebola Virus VP40 Modulates Cell Cycle and Biogenesis of Extracellular Vesicles.

Removal of HIV DNA by CRISPR from Patient Blood Engrafts in Humanized Mice.

Publisher Correction: Antiretroviral Drugs Alter the Content of Extracellular Vesicles from HIV-1-Infected Cells.

Complementary Mechanisms Potentially Involved in the Pathology of Zika Virus.

Autophagy, EVs, and Infections: A Perfect Question for a Perfect Time.

Toll-like receptor 3 regulates Zika virus infection and associated host inflammatory response in primary human astrocytes.

Morphine counteracts the antiviral effect of antiretroviral drugs and causes upregulation of p62/SQSTM1 and histone-modifying enzymes in HIV-infected astrocytes.

Effect of transcription inhibition and generation of suppressive viral non-coding RNAs.

Extracellular Vesicles and Ebola Virus: A New Mechanism of Immune Evasion.

Stem Cell Extracellular Vesicles and their Potential to Contribute to the Repair of Damaged CNS Cells.

An Omics Approach to Extracellular Vesicles from HIV-1 Infected Cells.