Thomas Brodnicki

A susceptibility allele from a non-diabetes-prone mouse strain accelerates diabetes in NOD congenic mice.

IL-1 receptor deficiency slows progression to diabetes in the NOD mouse.

Proapoptotic BH3-only Bcl-2 family member Bik/Blk/Nbk is expressed in hemopoietic and endothelial cells but is redundant for their programmed death.

Harp (harmonin-interacting, ankyrin repeat-containing protein), a novel protein that interacts with harmonin in epithelial tissues.

Fms-like tyrosine kinase 3 ligand administration overcomes a genetically determined dendritic cell deficiency in NOD mice and protects against diabetes development.

Localization of Idd11 is not associated with thymus and nkt cell abnormalities in NOD mice.

Congenic nonobese diabetic mouse strains fail to confirm linkage of a marginal zone B lymphocyte phenotype to the Idd11 locus on chromosome 4.

Mind the gap: analysis of marker-assisted breeding strategies for inbred mouse strains.

Monocytic leukemia zinc finger protein is essential for the development of long-term reconstituting hematopoietic stem cells.

Gene structure and transcript analysis of the human and mouse EGF-TM7 molecule, FIRE.

Two genetic loci independently confer susceptibility to autoimmune gastritis.

Somatic mutation and autoimmunity.

Insulin teaches a new lesson in tolerance.

An NZW-derived interval on chromosome 7 moderates sialadenitis, but not insulitis in congenic nonobese diabetic mice.

Recent insights into CD4+ T-cell specificity and function in type 1 diabetes.

Congenic mouse strains enable discrimination of genetic determinants contributing to fear and fear memory.

A recombination hotspot leads to sequence variability within a novel gene (AK005651) and contributes to type 1 diabetes susceptibility.

Generation of stable pluripotent stem cells from NOD mouse tail-tip fibroblasts.

The pro-apoptotic BH3-only protein Bid is dispensable for development of insulitis and diabetes in the non-obese diabetic mouse.

Audiogenic seizure proneness requires the contribution of two susceptibility loci in mice.