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Physics for Medicine Paris,
ESPCI Paris,
Inserm,
CNRS,
Physics for Medicine Paris, ESPCI Paris, INSERM, CNRS
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Phosphatidylinositol 3-kinase is a key mediator of central sensitization in painful inflammatory conditions.
The Journal of neuroscience : the official journal of the Society for Neuroscience Apr, 2008 | Pubmed ID: 18417706
BDNF-dependent plasticity induced by peripheral inflammation in the primary sensory and the cingulate cortex triggers cold allodynia and reveals a major role for endogenous BDNF as a tuner of the affective aspect of pain.
The Journal of neuroscience : the official journal of the Society for Neuroscience Oct, 2014 | Pubmed ID: 25355226
Transcranial functional ultrasound imaging of the brain using microbubble-enhanced ultrasensitive Doppler.
NeuroImage Jan, 2016 | Pubmed ID: 26416649
Ultrafast ultrasound localization microscopy for deep super-resolution vascular imaging.
Nature Nov, 2015 | Pubmed ID: 26607546
Orofacial Neuropathic Pain Leads to a Hyporesponsive Barrel Cortex with Enhanced Structural Synaptic Plasticity.
PloS one , 2016 | Pubmed ID: 27548330
Transcranial Functional Ultrasound Imaging in Freely Moving Awake Mice and Anesthetized Young Rats without Contrast Agent.
Ultrasound in medicine & biology 08, 2017 | Pubmed ID: 28476311
In Vivo Imaging of Single Tumor Cells in Fast-Flowing Bloodstream Using Near-Infrared Quantum Dots and Time-Gated Imaging.
ACS nano 03, 2019 | Pubmed ID: 30835434
4D functional ultrasound imaging of whole-brain activity in rodents.
Nature methods 10, 2019 | Pubmed ID: 31548704
Ultrafast ultrasound imaging pattern analysis reveals distinctive dynamic brain states and potent sub-network alterations in arthritic animals.
Scientific reports 06, 2020 | Pubmed ID: 32591574
Pharmaco-fUS: Quantification of pharmacologically-induced dynamic changes in brain perfusion and connectivity by functional ultrasound imaging in awake mice.
NeuroImage 11, 2020 | Pubmed ID: 32795659
Large-scale functional ultrasound imaging of the spinal cord reveals in-depth spatiotemporal responses of spinal nociceptive circuits in both normal and inflammatory states.
Pain Sep, 2020 | Pubmed ID: 32947542
PSL Research University
Adrien Bertolo*,1,
Mohamed Nouhoum*,1,
Silvia Cazzanelli*,1,
Jeremy Ferrier*,3,
Jean-Charles Mariani2,
Andrea Kliewer4,2,
Benoit Belliard1,
Bruno-Félix Osmanski3,
Thomas Deffieux*,1,
Sophie Pezet*,1,
Zsolt Lenkei*,2,
Mickael Tanter*,1
1Physics for Medicine Paris, ESPCI Paris, INSERM, CNRS, PSL Research University,
2Institute of Psychiatry and Neurosciences of Paris, INSERM U1266, Université de Paris,
3, Iconeus,
4Department of Pharmacology and Toxicology, Jena University Hospital - Friedrich Schiller University Jena
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