Christopher Fang-Yen

Distinct Mechanisms Underlie Quiescence during Two Caenorhabditis elegans Sleep-Like States.

Biomechanical analysis of gait adaptation in the nematode Caenorhabditis elegans.

Optogenetic manipulation of neural activity in freely moving Caenorhabditis elegans.

High-speed synthetic aperture microscopy for live cell imaging.

Overcoming the diffraction limit using multiple light scattering in a highly disordered medium.

Laser microsurgery in Caenorhabditis elegans.

IRK-1 potassium channels mediate peptidergic inhibition of Caenorhabditis elegans serotonin neurons via a G(o) signaling pathway.

Proprioceptive coupling within motor neurons drives C. elegans forward locomotion.

Scanner-free and wide-field endoscopic imaging by using a single multimode optical fiber.

Long-term imaging of Caenorhabditis elegans using nanoparticle-mediated immobilization.

Efficient single-cell transgene induction in Caenorhabditis elegans using a pulsed infrared laser.

Multi-well imaging of development and behavior in Caenorhabditis elegans.

Construction of a system for single-cell transgene induction in Caenorhabditis elegans using a pulsed infrared laser.

Neural and genetic degeneracy underlies Caenorhabditis elegans feeding behavior.

FMRFamide-like FLP-13 neuropeptides promote quiescence following heat stress in Caenorhabditis elegans.

Preferential coupling of an incident wave to reflection eigenchannels of disordered media.

Illuminating neural circuits and behaviour in Caenorhabditis elegans with optogenetics.

Altered Right Ventricular Kinetic Energy Work Density and Viscous Energy Dissipation in Patients with Pulmonary Arterial Hypertension: A Pilot Study Using 4D Flow MRI.

Simultaneous Optogenetic Stimulation of Individual Pharyngeal Neurons and Monitoring of Feeding Behavior in Intact C. elegans.

An Imaging System for C. elegans Behavior.

Evidence for Novel Pharmacological Sensitivities of Transient Receptor Potential (TRP) Channels in Schistosoma mansoni.

Pharyngeal pumping in Caenorhabditis elegans depends on tonic and phasic signaling from the nervous system.

Ageing with elegans: a research proposal to map healthspan pathways.

The RFamide receptor DMSR-1 regulates stress-induced sleep in .

Food responsiveness regulates episodic behavioral states in .

Quantitative Assessment of Fat Levels in Using Dark Field Microscopy.

Longitudinal imaging of in a microfabricated device reveals variation in behavioral decline during aging.

Antagonistic Serotonergic and Octopaminergic Neural Circuits Mediate Food-Dependent Locomotory Behavior in .

Comparing Caenorhabditis elegans gentle and harsh touch response behavior using a multiplexed hydraulic microfluidic device.

Excitatory motor neurons are local oscillators for backward locomotion.

Distributed rhythm generators underlie forward locomotion.

A sleep state in larvae required for neural stem cell proliferation.

Quantitative imaging of sleep behavior in Caenorhabditis elegans and larval Drosophila melanogaster.

Optimized criteria for locomotion-based healthspan evaluation in C. elegans using the WorMotel system.

A quiescent state following mild sensory arousal in Caenorhabditis elegans is potentiated by stress.

Dehydrated Stocks Are Resistant to Multiple Freeze-Thaw Cycles.

Preconditioning of Caenorhabditis elegans to anoxic insult by inactivation of cholinergic, GABAergic and muscle activity.

Sleep Induction by Mechanosensory Stimulation in Drosophila.

Thermal laser ablation with tunable lesion size reveals multiple origins of seizure-like convulsions in Caenorhabditis elegans.

DAF-16/FoxO and DAF-12/VDR control cellular plasticity both cell-autonomously and via interorgan signaling.

Phase response analyses support a relaxation oscillator model of locomotor rhythm generation in .

Cryopreservation of dehydrated with multiple recoveries using a granular medium.

Optogenetic Perturbation of Individual C. elegans Pharyngeal Neurons While Monitoring Feeding Behavior.

An Imaging System for Monitoring C. elegans Behavior and Aging.

Pharyngeal timing and particle transport defects in feeding mutants.