Hendrik Wildner

The bHLH factor Olig3 coordinates the specification of dorsal neurons in the spinal cord.

The homeodomain transcription factor Gbx1 identifies a subpopulation of late-born GABAergic interneurons in the developing dorsal spinal cord.

dILA neurons in the dorsal spinal cord are the product of terminal and non-terminal asymmetric progenitor cell divisions, and require Mash1 for their development.

Postnatal loss of Dlk1 imprinting in stem cells and niche astrocytes regulates neurogenesis.

Fast synaptic inhibition in spinal sensory processing and pain control.

Ascl1 (Mash1) knockout perturbs differentiation of nonneuronal cells in olfactory epithelium.

Genome-wide expression analysis of Ptf1a- and Ascl1-deficient mice reveals new markers for distinct dorsal horn interneuron populations contributing to nociceptive reflex plasticity.

Morphological, biophysical and synaptic properties of glutamatergic neurons of the mouse spinal dorsal horn.

Binary recombinase systems for high-resolution conditional mutagenesis.

A subcortical inhibitory signal for behavioral arrest in the thalamus.

Targeted ablation, silencing, and activation establish glycinergic dorsal horn neurons as key components of a spinal gate for pain and itch.

Several posttranslational modifications act in concert to regulate gephyrin scaffolding and GABAergic transmission.

Spinal nociceptive circuit analysis with recombinant adeno-associated viruses: the impact of serotypes and promoters.

Identification of Two Classes of Somatosensory Neurons That Display Resistance to Retrograde Infection by Rabies Virus.

Immune or Genetic-Mediated Disruption of CASPR2 Causes Pain Hypersensitivity Due to Enhanced Primary Afferent Excitability.

Circuit dissection of the role of somatostatin in itch and pain.

Author Correction: Circuit dissection of the role of somatostatin in itch and pain.