Wang Wang

Alteration of vascular response to norepinephrine, calcitonin gene-related peptide, and acetylcholine in orchidectomized rats.

Imaging microdomain Ca2+ in muscle cells.

Intramuscular gene transfer of CGRP inhibits neointimal hyperplasia after balloon injury in the rat abdominal aorta.

Sustained beta1-adrenergic stimulation modulates cardiac contractility by Ca2+/calmodulin kinase signaling pathway.

Phosphatidylinositol 3-kinase offsets cAMP-mediated positive inotropic effect via inhibiting Ca2+ influx in cardiomyocytes.

ASF/SF2-regulated CaMKIIdelta alternative splicing temporally reprograms excitation-contraction coupling in cardiac muscle.

Endogenous calcitonin gene-related peptide protects human alveolar epithelial cells through protein kinase Cepsilon and heat shock protein.

Nitroxyl improves cellular heart function by directly enhancing cardiac sarcoplasmic reticulum Ca2+ cycling.

Ellagic acid from acorn fringe by enzymatic hydrolysis and combined effects of operational variables and enzymes on yield of the production.

Parvalbumin isoforms differentially accelerate cardiac myocyte relaxation kinetics in an animal model of diastolic dysfunction.

Novel anti-oxidative role of calreticulin in protecting A549 human type II alveolar epithelial cells against hypoxic injury.

Cardiac-directed parvalbumin transgene expression in mice shows marked heart rate dependence of delayed Ca2+ buffering action.

Transforming growth factor beta induces apoptosis through repressing the phosphoinositide 3-kinase/AKT/survivin pathway in colon cancer cells.

Parvalbumin isoforms for enhancing cardiac diastolic function.

Superoxide flashes in single mitochondria.

Designing heart performance by gene transfer.

Artificial selection for whole animal low intrinsic aerobic capacity co-segregates with hypoxia-induced cardiac pump failure.

Involvement of the post-transcriptional regulator Hfq in Yersinia pestis virulence.

Superoxide flashes: illuminating new insights into cardiac ischemia/reperfusion injury.

Molecular cardiology in translation: gene, cell and chemical-based experimental therapeutics for the failing heart.

Parvalbumin: Targeting calcium handling in cardiac diastolic dysfunction.

[Evaluation of immunization protection efficacy of plague subunit vaccine].

Different strategies for preparation of non-tagged rV270 protein and its efficacy against Yersinia pestis challenge.

Prevention of angiotensin II-mediated renal oxidative stress, inflammation, and fibrosis by angiotensin-converting enzyme 2.

Enhanced susceptibility to biomechanical stress in ACE2 null mice is prevented by loss of the p47(phox) NADPH oxidase subunit.

Use of ginseng to reduce post-myocardial adverse myocardial remodeling: applying scientific principles to the use of herbal therapies.

Imaging superoxide flash and metabolism-coupled mitochondrial permeability transition in living animals.

Role of ACE2 in diastolic and systolic heart failure.

Response to "A critical evaluation of cpYFP as a probe for superoxide".

Superoxide flashes reveal novel properties of mitochondrial reactive oxygen species excitability in cardiomyocytes.

Loss of angiotensin-converting enzyme-2 exacerbates diabetic cardiovascular complications and leads to systolic and vascular dysfunction: a critical role of the angiotensin II/AT1 receptor axis.

Cardioprotective effects mediated by angiotensin II type 1 receptor blockade and enhancing angiotensin 1-7 in experimental heart failure in angiotensin-converting enzyme 2-null mice.

Targeting the ACE2 and Apelin Pathways Are Novel Therapies for Heart Failure: Opportunities and Challenges.

Calcium mishandling in diastolic dysfunction: mechanisms and potential therapies.

N-Phenyl-4-hydroxy-2-quinolone-3-carboxamides as selective inhibitors of mutant H1047R phosphoinositide-3-kinase (PI3Kα).

Enhanced recovery from ischemia-reperfusion injury in PI3Kα dominant negative hearts: investigating the role of alternate PI3K isoforms, increased glucose oxidation and MAPK signaling.

Wnt5a inhibits hypoxia-induced pulmonary arterial smooth muscle cell proliferation by downregulation of β-catenin.

Noncanonical EF-hand motif strategically delays Ca(2+) buffering to enhance cardiac performance.

Respective contribution of mitochondrial superoxide and pH to mitochondria-targeted circularly permuted yellow fluorescent protein (mt-cpYFP) flash activity.

Loss of p47phox subunit enhances susceptibility to biomechanical stress and heart failure because of dysregulation of cortactin and actin filaments.

Loss of Apelin exacerbates myocardial infarction adverse remodeling and ischemia-reperfusion injury: therapeutic potential of synthetic Apelin analogues.

Mitochondrial complex I deficiency increases protein acetylation and accelerates heart failure.

[Study on human aFGF fusion gene transformation with soybean 24 kDa oleosin and expression in safflower].

[Prognostic factors and outcomes of Legg-Calvé-Perthes disease].