Kristian Riesbeck

Ciprofloxacin induces an immunomodulatory stress response in human T lymphocytes.

Endothelial cells expressing an inflammatory phenotype are lysed by superantigen-targeted cytotoxic T cells.

Destructive knee joint infection caused by Peptostreptococcus micros: importance of early microbiological diagnosis.

Long-term persistence of a discotheque-associated invasive Neisseria meningitidis group C strain as proven by pulsed-field gel electrophoresis and porA gene sequencing.

Susceptibilities of Mycobacterium marinum to gatifloxacin, gemifloxacin, levofloxacin, linezolid, moxifloxacin, telithromycin, and quinupristin-dalfopristin (Synercid) compared to its susceptibilities to reference macrolides and quinolones.

The novel IgD binding protein from Moraxella catarrhalis induces human B lymphocyte activation and Ig secretion in the presence of Th2 cytokines.

The immunoglobulin D-binding part of the outer membrane protein MID from Moraxella catarrhalis comprises 238 amino acids and a tetrameric structure.

CTLA4Ig combined with anti-LFA-1 prolongs cardiac allograft survival indefinitely.

A pilot with pain in his leg: thigh abscess caused by Salmonella enterica serotype Brandenburg.

Comparison of the antibacterial activities of ampicillin, ciprofloxacin, clarithromycin, telithromycin and quinupristin/dalfopristin against intracellular non-typeable Haemophilus influenzae.

The Moraxella catarrhalis immunoglobulin D-binding protein MID has conserved sequences and is regulated by a mechanism corresponding to phase variation.

Nontypeable Haemophilus influenzae activates human eosinophils through beta-glucan receptors.

The immunoglobulin D-binding protein MID from Moraxella catarrhalis is also an adhesin.

Several gene programs are induced in ciprofloxacin-treated human lymphocytes as revealed by microarray analysis.

Paronychia due to Prevotella bivia that resulted in amputation: fast and correct bacteriological diagnosis is crucial.

Dicloxacillin and erythromycin at high concentrations increase ICAM-1 expression by endothelial cells: a possible factor in the pathogenesis of infusion phlebitis.

Immunization with the truncated adhesin moraxella catarrhalis immunoglobulin D-binding protein (MID764-913) is protective against M. catarrhalis in a mouse model of pulmonary clearance.

A novel localization of the G-protein-coupled CysLT1 receptor in the nucleus of colorectal adenocarcinoma cells.

The respiratory pathogen Moraxella catarrhalis adheres to epithelial cells by interacting with fibronectin through ubiquitous surface proteins A1 and A2.

Ionic binding of C3 to the human pathogen Moraxella catarrhalis is a unique mechanism for combating innate immunity.

The IgD-binding domain of the Moraxella IgD-binding protein MID (MID962-1200) activates human B cells in the presence of T cell cytokines.

Haemophilus influenzae surface fibrils contribute to serum resistance by interacting with vitronectin.

Streptococcal M protein: a multipotent and powerful inducer of inflammation.

The respiratory pathogen moraxella catarrhalis binds to laminin via ubiquitous surface proteins A1 and A2.

The IgD CH1 region contains the binding site for the human respiratory pathogen Moraxella catarrhalis IgD-binding protein MID.

Wnt-5a mRNA translation is suppressed by the Elav-like protein HuR in human breast epithelial cells.

MID and UspA1/A2 of the human respiratory pathogen Moraxella catarrhalis, and interactions with the human host as basis for vaccine development.

Comparison of the serological responses to Moraxella catarrhalis immunoglobulin D-binding outer membrane protein and the ubiquitous surface proteins A1 and A2.

Immunomodulation by fluoroquinolones and other antibacterial agents.

Purification of IgD from human serum--a novel application of recombinant M. catarrhalis IgD-binding protein (MID).

Structure and immunological action of the human pathogen Moraxella catarrhalis IgD-binding protein.

Haemophilus influenzae survival during complement-mediated attacks is promoted by Moraxella catarrhalis outer membrane vesicles.

Characterization of the IgD binding site of encapsulated Haemophilus influenzae serotype b.

Interaction with C4b-binding protein contributes to nontypeable Haemophilus influenzae serum resistance.

Biphasic effect of gingipains from Porphyromonas gingivalis on the human complement system.

Comparison of an automated Borrelia indirect chemiluminescent immunoassay (CLIA) with a VlsE/C6 ELISA and Immunoblot.

Identification of a novel Haemophilus influenzae protein important for adhesion to epithelial cells.

Moraxella-dependent alpha 1-antichymotrypsin neutralization: a unique virulence mechanism.

Protein D of Haemophilus influenzae: a protective nontypeable H. influenzae antigen and a carrier for pneumococcal conjugate vaccines.

Purification of alpha1-antichymotrypsin from human plasma with recombinant M. catarrhalis ubiquitous surface protein A1.

Moraxella catarrhalis-dependent tonsillar B cell activation does not lead to apoptosis but to vigorous proliferation resulting in nonspecific IgM production.

Current progress of adhesins as vaccine candidates for Moraxella catarrhalis.

The Moraxella IgD-binding protein MID/Hag is an oligomeric autotransporter.

Haemophilus influenzae interacts with the human complement inhibitor factor H.

Binding of complement inhibitor C4b-binding protein contributes to serum resistance of Porphyromonas gingivalis.

Nontypeable Haemophilus influenzae adhesin protein E: characterization and biological activity.

Interpain A, a cysteine proteinase from Prevotella intermedia, inhibits complement by degrading complement factor C3.

Superantigen- and TLR-dependent activation of tonsillar B cells after receptor-mediated endocytosis.

Virulence mechanisms of Moraxella in the pathogenesis of infection.

Complement evasion strategies of pathogens-acquisition of inhibitors and beyond.

Clinical isolates of Streptococcus pneumoniae bind the complement inhibitor C4b-binding protein in a PspC allele-dependent fashion.

Immunoglobulin D enhances immune surveillance by activating antimicrobial, proinflammatory and B cell-stimulating programs in basophils.

Nontypeable Haemophilus influenzae protein E binds vitronectin and is important for serum resistance.

Protein E of Haemophilus influenzae is a ubiquitous highly conserved adhesin.

Binding of complement regulators to invasive nontypeable Haemophilus influenzae isolates is not increased compared to nasopharyngeal isolates, but serum resistance is linked to disease severity.

B cell activation by outer membrane vesicles--a novel virulence mechanism.

Vitronectin binds to the head region of Moraxella catarrhalis ubiquitous surface protein A2 and confers complement-inhibitory activity.

Haemophilus influenzae and the complement system.

Genome analysis of Moraxella catarrhalis strain BBH18, [corrected] a human respiratory tract pathogen.

Vitronectin in bacterial pathogenesis: a host protein used in complement escape and cellular invasion.

Multicomponent Moraxella catarrhalis outer membrane vesicles induce an inflammatory response and are internalized by human epithelial cells.

Bacterial outer membrane vesicles in disease and preventive medicine.

Immune evasion of Moraxella catarrhalis involves ubiquitous surface protein A-dependent C3d binding.

Nucleotide-binding and oligomerization domain-like receptors and retinoic acid inducible gene-like receptors in human tonsillar T lymphocytes.

Comparative study of immune status to infectious agents in elderly patients with multiple myeloma, Waldenstrom's macroglobulinemia, and monoclonal gammopathy of undetermined significance.

Haemophilus influenzae protein E recognizes the C-terminal domain of vitronectin and modulates the membrane attack complex.

Moraxella catarrhalis outer membrane vesicles carry β-lactamase and promote survival of Streptococcus pneumoniae and Haemophilus influenzae by inactivating amoxicillin.

A descriptive study of bacterial load of full-thickness surgical wounds in dermatologic surgery.

Necrotizing myositis and septic shock caused by Haemophilus influenzae type f in a previously healthy man diagnosed with an IgG3 and a mannose-binding lectin deficiency.

Haemophilus influenzae protein E binds to the extracellular matrix by concurrently interacting with laminin and vitronectin.

A role for TLRs in Moraxella-superantigen induced polyclonal B cell activation.

A metalloproteinase karilysin present in the majority of Tannerella forsythia isolates inhibits all pathways of the complement system.

Crystallization and X-ray diffraction analysis of a novel surface-adhesin protein: protein E from Haemophilus influenzae.

Acquisition of complement inhibitor serine protease factor I and its cofactors C4b-binding protein and factor H by Prevotella intermedia.

Human pathogens utilize host extracellular matrix proteins laminin and collagen for adhesion and invasion of the host.

Comparative analysis of the humoral immune response to Moraxella catarrhalis and Streptococcus pneumoniae surface antigens in children suffering from recurrent acute otitis media and chronic otitis media with effusion.

Increase of β-lactam-resistant invasive Haemophilus influenzae in Sweden, 1997 to 2010.

Enolase of Streptococcus pneumoniae binds human complement inhibitor C4b-binding protein and contributes to complement evasion.

Moraxella catarrhalis: from interactions with the host immune system to vaccine development.

QseC controls biofilm formation of non-typeable Haemophilus influenzae in addition to an AI-2-dependent mechanism.

Outer membrane vesicles shield Moraxella catarrhalis β-lactamase from neutralization by serum IgG.

Genome sequence of Moraxella catarrhalis RH4, an isolate of seroresistant lineage.

The activation pattern of blood leukocytes in head and neck squamous cell carcinoma is correlated to survival.

The unique structure of Haemophilus influenzae protein E reveals multiple binding sites for host factors.

Streptococcus pneumoniae endopeptidase O (PepO) is a multifunctional plasminogen- and fibronectin-binding protein, facilitating evasion of innate immunity and invasion of host cells.

Haemophilus influenzae acquires vitronectin via the ubiquitous Protein F to subvert host innate immunity.

Impact of sequence diversity in the Moraxella catarrhalis UspA2/UspA2H head domain on vitronectin binding and antigenic variation.

The choline-binding protein PspC of Streptococcus pneumoniae interacts with the C-terminal heparin-binding domain of vitronectin.

Group A streptococci are protected from amoxicillin-mediated killing by vesicles containing β-lactamase derived from Haemophilus influenzae.

An alternative role of C1q in bacterial infections: facilitating Streptococcus pneumoniae adherence and invasion of host cells.

Complete Genome Sequence of Encapsulated Haemophilus influenzae Type f KR494, an Invasive Isolate That Caused Necrotizing Myositis.

High incidence of septic shock caused by Streptococcus pneumoniae serotype 3--a retrospective epidemiological study.

Haemophilus influenzae resides in tonsils and uses immunoglobulin D binding as an evasion strategy.

Prevalence, distribution and transfer of small β-lactamase-containing plasmids in Swedish Haemophilus influenzae.

Comparative genomic analysis reveals distinct genotypic features of the emerging pathogen Haemophilus influenzae type f.

Diversion of the host humoral response: a novel virulence mechanism of Haemophilus influenzae mediated via outer membrane vesicles.

Candida albicans uses the surface protein Gpm1 to attach to human endothelial cells and to keratinocytes via the adhesive protein vitronectin.

Impact of immunization with Protein F on pulmonary clearance of nontypeable Haemophilus influenzae.

A fine-tuned interaction between trimeric autotransporter haemophilus surface fibrils and vitronectin leads to serum resistance and adherence to respiratory epithelial cells.

In vitro selection of RNA aptamers directed against protein E: a Haemophilus influenzae adhesin.

Haemophilus influenzae: recent advances in the understanding of molecular pathogenesis and polymicrobial infections.

Risk factors for pneumococcal carriage in day care centers: a retrospective study during a 10-year period.

Binding of Streptococcus pneumoniae endopeptidase O (PepO) to complement component C1q modulates the complement attack and promotes host cell adherence.

Outer membrane protein OlpA contributes to Moraxella catarrhalis serum resistance via interaction with factor H and the alternative pathway.

Identification of a Haemophilus influenzae factor H-Binding lipoprotein involved in serum resistance.

Haemophilus influenzae stores and distributes hemin by using protein E.

Streptococcus pneumoniae phosphoglycerate kinase is a novel complement inhibitor affecting the membrane attack complex formation.

Haemophilus influenzae surface fibril (Hsf) is a unique twisted hairpin-like trimeric autotransporter.

Prevalence of penicillin-non-susceptible Streptococcus pneumoniae in children in day-care centres subjected to an intervention to prevent dispersion.

Collagen VI Is Upregulated in COPD and Serves Both as an Adhesive Target and a Bactericidal Barrier for Moraxella catarrhalis.

Identification of Haemophilus influenzae Type b Isolates by Use of Matrix-Assisted Laser Desorption Ionization-Time of Flight Mass Spectrometry.

A fusion protein derived from Moraxella catarrhalis and Neisseria meningitidis aimed for immune modulation of human B cells.

Moraxella catarrhalis induces an immune response in the murine lung that is independent of human CEACAM5 expression and long-term smoke exposure.

Identification of outer membrane Porin D as a vitronectin-binding factor in cystic fibrosis clinical isolates of Pseudomonas aeruginosa.

Moraxella catarrhalis Binds Plasminogen To Evade Host Innate Immunity.

Haemophilus influenzae P4 Interacts With Extracellular Matrix Proteins Promoting Adhesion and Serum Resistance.

A Metalloproteinase Mirolysin of Tannerella forsythia Inhibits All Pathways of the Complement System.

Pseudomonas aeruginosa Uses Dihydrolipoamide Dehydrogenase (Lpd) to Bind to the Human Terminal Pathway Regulators Vitronectin and Clusterin to Inhibit Terminal Pathway Complement Attack.

Haemophilus influenzae Type f Hijacks Vitronectin Using Protein H To Resist Host Innate Immunity and Adhere to Pulmonary Epithelial Cells.

Outbreak of a beta-lactam resistant non-typeable Haemophilus influenzae sequence type 14 associated with severe clinical outcomes.

Moraxella catarrhalis Evades Host Innate Immunity via Targeting Cartilage Oligomeric Matrix Protein.

Conserved Patterns of Microbial Immune Escape: Pathogenic Microbes of Diverse Origin Target the Human Terminal Complement Inhibitor Vitronectin via a Single Common Motif.

The Respiratory Pathogen Moraxella catarrhalis Targets Collagen for Maximal Adherence to Host Tissues.

Moonlighting of Helicobacter pylori catalase protects against complement-mediated killing by utilising the host molecule vitronectin.

The prevalence, population structure and screening test specificity of penicillin-susceptible Staphylococcus aureus bacteremia isolates in Malmö, Sweden.

Host-pathogen interactions of nontypeable Haemophilus influenzae: from commensal to pathogen.

Reduction of Streptococcus pneumoniae in upper respiratory tract cultures and a decreased incidence of related acute otitis media following introduction of childhood pneumococcal conjugate vaccines in a Swedish county.

FACIN, a Double-Edged Sword of the Emerging Periodontal Pathogen Filifactor alocis: A Metabolic Enzyme Moonlighting as a Complement Inhibitor.

Can dressings soaked with polyhexanide reduce bacterial loads in full-thickness skin grafting? A randomized controlled trial.

Inhaled Corticosteroids in Chronic Obstructive Pulmonary Disease. A Two-Edged Sword.

PRELP Enhances Host Innate Immunity against the Respiratory Tract Pathogen .

The crystal structure of PD1, a Haemophilus surface fibril domain.

The Adr1 Interacts with the C-Terminus of Human Vitronectin in a Salt-Sensitive Manner.

Fluoroquinolone-Resistant Alcaligenes faecalis Related to Chronic Suppurative Otitis Media, Angola.

Haemophilus Protein F Orthologs of Pathogens Infecting the Airways: Exploiting Host Laminin at Heparin-Binding Sites for Maximal Adherence to Epithelial Cells.

Hemolytic Uremic Syndrome Associated With Pneumococci in Children-An Elusive Mystery Now Explained?

Genetic predisposition to infection in a case of atypical hemolytic uremic syndrome.

Capsule Typing of Haemophilus influenzae by Matrix-Assisted Laser Desorption/Ionization Time-of-Flight Mass Spectrometry.

Aerobic bacteria associated with chronic suppurative otitis media in Angola.

Titanium granules pre-treated with hydrogen peroxide inhibit growth of bacteria associated with post-operative infections in spine surgery.

A novel PBP3 substitution in Haemophilus influenzae confers reduced aminopenicillin susceptibility.

Update on non-typeable Haemophilus influenzae-mediated disease and vaccine development.

How bacteria hack the matrix and dodge the bullets of immunity.

Benzylpenicillin versus wide-spectrum beta-lactam antibiotics as empirical treatment of Haemophilus influenzae-associated lower respiratory tract infections in adults; a retrospective propensity score-matched study.

Highlights of the São Paulo ISEV workshop on extracellular vesicles in cross-kingdom communication.

Bacterial Outer Membrane Vesicles Induce Vitronectin Release Into the Bronchoalveolar Space Conferring Protection From Complement-Mediated Killing.

The Pulmonary Extracellular Matrix Is a Bactericidal Barrier Against in Chronic Obstructive Pulmonary Disease (COPD): Implications for an Innate Host Defense Function of Collagen VI.

Outer Membrane Vesicles Protect the Pathogen From Reactive Oxygen Species of the Respiratory Burst.

Short Leucine-Rich Proteoglycans Modulate Complement Activity and Increase Killing of the Respiratory Pathogen .