Niels Voigt

DNA-programmed control of photosensitized singlet oxygen production.

Differential phosphorylation-dependent regulation of constitutively active and muscarinic receptor-activated IK,ACh channels in patients with chronic atrial fibrillation.

Changes in I K, ACh single-channel activity with atrial tachycardia remodelling in canine atrial cardiomyocytes.

Inhibition of IK,ACh current may contribute to clinical efficacy of class I and class III antiarrhythmic drugs in patients with atrial fibrillation.

Single-molecule chemical reactions on DNA origami.

Left-to-right atrial inward rectifier potassium current gradients in patients with paroxysmal versus chronic atrial fibrillation.

Multiple potential molecular contributors to atrial hypocontractility caused by atrial tachycardia remodeling in dogs.

The ryanodine receptor channel as a molecular motif in atrial fibrillation: pathophysiological and therapeutic implications.

Voltage-clamp-based methods for the detection of constitutively active acetylcholine-gated I(K,ACh) channels in the diseased heart.

Functional patterning of DNA origami by parallel enzymatic modification.

A DNA tile actuator with eleven discrete states.

Ca(2+)-related signaling and protein phosphorylation abnormalities play central roles in a new experimental model of electrical storm.

DNA origami: a quantum leap for self-assembly of complex structures.

Oxidized CaMKII causes cardiac sinus node dysfunction in mice.

Recent advances in the molecular pathophysiology of atrial fibrillation.

Defects in ankyrin-based membrane protein targeting pathways underlie atrial fibrillation.

Differential protein kinase C isoform regulation and increased constitutive activity of acetylcholine-regulated potassium channels in atrial remodeling.

Human atrial action potential and Ca2+ model: sinus rhythm and chronic atrial fibrillation.

DNA-programmed Glaser-Eglinton reactions for the synthesis of conjugated molecular wires.

Inhibition of CaMKII Phosphorylation of RyR2 Prevents Induction of Atrial Fibrillation in FKBP12.6 Knockout Mice.

Multilayer DNA origami packed on hexagonal and hybrid lattices.

Proarrhythmic atrial calcium cycling in the diseased heart.

Enhanced sarcoplasmic reticulum Ca2+ leak and increased Na+-Ca2+ exchanger function underlie delayed afterdepolarizations in patients with chronic atrial fibrillation.

Role of RyR2 phosphorylation at S2814 during heart failure progression.

Calcium handling and atrial fibrillation.

Transient receptor potential canonical-3 channel-dependent fibroblast regulation in atrial fibrillation.

Cellular and molecular correlates of ectopic activity in patients with atrial fibrillation.

The biology of human pulmonary veins: does it help us to better understand AF pathophysiology in patients?

New directions in antiarrhythmic drug therapy for atrial fibrillation.

MicroRNA29: a mechanistic contributor and potential biomarker in atrial fibrillation.

Impaired Na(+)-dependent regulation of acetylcholine-activated inward-rectifier K(+) current modulates action potential rate dependence in patients with chronic atrial fibrillation.

Finding Ms or Mr Right: Which miRNA to target in AF?

Nucleoside Diphosphate Kinase-C Suppresses cAMP Formation in Human Heart Failure.

Inverse remodelling of K2P3.1 K+ channel expression and action potential duration in left ventricular dysfunction and atrial fibrillation: implications for patient-specific antiarrhythmic drug therapy.

Stretch-activated two-pore-domain (K) potassium channels in the heart: Focus on atrial fibrillation and heart failure.

The inward rectifier current inhibitor PA-6 terminates atrial fibrillation and does not cause ventricular arrhythmias in goat and dog models.

In search for novel functions of adenosine 5'-triphosphate (ATP) in the heart.

Report on the Ion Channel Symposium : Organized by the German Cardiac Society Working Group on Cellular Electrophysiology (AG 18).

Atrial fibrillation and heart failure-associated remodeling of two-pore-domain potassium (K) channels in murine disease models: focus on TASK-1.

German Cardiac Society Working Group on Cellular Electrophysiology state-of-the-art paper: impact of molecular mechanisms on clinical arrhythmia management.

Axial Tubule Junctions Activate Atrial Ca Release Across Species.

ESC Congress 2018 highlights in basic science: a report from the Scientists of Tomorrow.

Ryanodine receptor dysfunction and the resolution revolution: how Nobel Prize-winning techniques transform cardiovascular research.

N-glycosylation-dependent regulation of hK17.1 currents.

Scientists on the Spot: Autophagy and heart disease.

CaMKII activity contributes to homeometric autoregulation of the heart: A novel mechanism for the Anrep effect.

Altered Atrial Cytosolic Calcium Handling Contributes to the Development of Postoperative Atrial Fibrillation.

Dysferlin links excitation-contraction coupling to structure and maintenance of the cardiac transverse-axial tubule system.

Insights into cardiovascular research in Göttingen and Heidelberg: a report by the ESC Scientists of Tomorrow.

Cellular and mitochondrial mechanisms of atrial fibrillation.