Nathalie Arbour

c-Jun NH(2)-terminal kinase (JNK)1 and JNK2 signaling pathways have divergent roles in CD8(+) T cell-mediated antiviral immunity.

Measles virus infects and suppresses proliferation of T lymphocytes from transgenic mice bearing human signaling lymphocytic activation molecule.

A new approach for evaluating antigen-specific T cell responses to myelin antigens during the course of multiple sclerosis.

Measles virus interacts with human SLAM receptor on dendritic cells to cause immunosuppression.

Distinctive properties of human adult brain-derived myelin progenitor cells.

Reduced endocannabinoid immune modulation by a common cannabinoid 2 (CB2) receptor gene polymorphism: possible risk for autoimmune disorders.

TLR signaling tailors innate immune responses in human microglia and astrocytes.

A new clinically relevant approach to expand myelin specific T cells.

Potential for interferon beta-induced serum antibodies in multiple sclerosis to inhibit endogenous interferon-regulated chemokine/cytokine responses within the central nervous system.

NKG2D-mediated cytotoxicity toward oligodendrocytes suggests a mechanism for tissue injury in multiple sclerosis.

Th1 polarization of CD4+ T cells by Toll-like receptor 3-activated human microglia.

Human TH17 lymphocytes promote blood-brain barrier disruption and central nervous system inflammation.

The blood-brain barrier induces differentiation of migrating monocytes into Th17-polarizing dendritic cells.

Activated leukocyte cell adhesion molecule promotes leukocyte trafficking into the central nervous system.

Dendritic cell differentiation signals induce anti-inflammatory properties in human adult microglia.

TGF-alpha as a candidate tumor antigen for renal cell carcinomas.

Preferential recruitment of interferon-gamma-expressing TH17 cells in multiple sclerosis.

Human activated T lymphocytes modulate IDO expression in tumors through Th1/Th2 balance.

Contribution of CD8 T lymphocytes to the immuno-pathogenesis of multiple sclerosis and its animal models.

Contribution of astrocyte-derived IL-15 to CD8 T cell effector functions in multiple sclerosis.

IL-27 increases the proliferation and effector functions of human naïve CD8+ T lymphocytes and promotes their development into Tc1 cells.

Endogenously expressed matrix protein M1 and nucleoprotein of influenza A are efficiently presented by class I and class II major histocompatibility complexes.

The majority of infiltrating CD8 T lymphocytes in multiple sclerosis lesions is insensitive to enhanced PD-L1 levels on CNS cells.

B cell-derived IL-15 enhances CD8 T cell cytotoxicity and is increased in multiple sclerosis patients.

Central nervous system recruitment of effector memory CD8+ T lymphocytes during neuroinflammation is dependent on α4 integrin.

Human brain endothelial cells endeavor to immunoregulate CD8 T cells via PD-1 ligand expression in multiple sclerosis.

Lipocalin 2 is a novel immune mediator of experimental autoimmune encephalomyelitis pathogenesis and is modulated in multiple sclerosis.

Journal club: Intrathecal effects of daclizumab treatment of multiple sclerosis.

Stimulation of Wnt/ß-catenin pathway in human CD8+ T lymphocytes from blood and lung tumors leads to a shared young/memory phenotype.

Melanoma cell adhesion molecule identifies encephalitogenic T lymphocytes and promotes their recruitment to the central nervous system.

Cytotoxic NKG2C+ CD4 T cells target oligodendrocytes in multiple sclerosis.

Diminished Th17 (not Th1) responses underlie multiple sclerosis disease abrogation after hematopoietic stem cell transplantation.

Mitochondrial damage revealed by immunoselection for ALS-linked misfolded SOD1.

Maraviroc and JC virus-associated immune reconstitution inflammatory syndrome.