Department of Child Health
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Ablation of D1 dopamine receptor-expressing cells generates mice with seizures, dystonia, hyperactivity, and impaired oral behavior.
Proceedings of the National Academy of Sciences of the United States of America Mar, 2007 | Pubmed ID: 17360497
Role of CCL2 (MCP-1) in traumatic brain injury (TBI): evidence from severe TBI patients and CCL2-/- mice.
Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism Apr, 2010 | Pubmed ID: 20029451
Involvement of pro- and anti-inflammatory cytokines and chemokines in the pathophysiology of traumatic brain injury.
Neurotherapeutics : the journal of the American Society for Experimental NeuroTherapeutics Jan, 2010 | Pubmed ID: 20129494
Deficiency of the chemokine receptor CXCR2 attenuates neutrophil infiltration and cortical damage following closed head injury.
Neurobiology of disease Nov, 2010 | Pubmed ID: 20621186
Kinin receptor antagonists as potential neuroprotective agents in central nervous system injury.
Molecules (Basel, Switzerland) Sep, 2010 | Pubmed ID: 20877247
Attenuated neurological deficit, cell death and lesion volume in Fas-mutant mice is associated with altered neuroinflammation following traumatic brain injury.
Brain research Sep, 2011 | Pubmed ID: 21871613
Rod microglia: elongation, alignment, and coupling to form trains across the somatosensory cortex after experimental diffuse brain injury.
Journal of neuroinflammation , 2012 | Pubmed ID: 23111107
Microglia: dismantling and rebuilding circuits after acute neurological injury.
Metabolic brain disease Apr, 2015 | Pubmed ID: 24733573
Rod microglia: a morphological definition.
PloS one , 2014 | Pubmed ID: 24830807
Commentary on Kamper et. al., juvenile traumatic brain injury evolves into a chronic brain disorder: The challenges in longitudinal studies of juvenile traumatic brain injury.
Experimental neurology Nov, 2014 | Pubmed ID: 24931226
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