M. Scott Bowers

Compound stimulus presentation and the norepinephrine reuptake inhibitor atomoxetine enhance long-term extinction of cocaine-seeking behavior.

The temporal sequence of changes in gene expression by drugs of abuse.

Forebrain astroglial plasticity is induced following withdrawal from repeated cocaine administration.

Activator of G protein signaling 3: a gatekeeper of cocaine sensitization and drug seeking.

Cocaine self-administration selectively abolishes LTD in the core of the nucleus accumbens.

Accumbens neurochemical adaptations produced by binge-like alcohol consumption.

Withdrawal from intermittent ethanol exposure increases probability of burst firing in VTA neurons in vitro.

Acamprosate attenuates cocaine- and cue-induced reinstatement of cocaine-seeking behavior in rats.

Cocaine but not natural reward self-administration nor passive cocaine infusion produces persistent LTP in the VTA.

Nucleus accumbens AGS3 expression drives ethanol seeking through G betagamma.

Reward-predictive cues enhance excitatory synaptic strength onto midbrain dopamine neurons.

Strain specific synaptic modifications on ventral tegmental area dopamine neurons after ethanol exposure.

Orexin A/hypocretin-1 selectively promotes motivation for positive reinforcers.

Reduced nucleus accumbens SK channel activity enhances alcohol seeking during abstinence.

AMPA receptor synaptic plasticity induced by psychostimulants: the past, present, and therapeutic future.

Conserved role of unc-79 in ethanol responses in lightweight mutant mice.

Exposure of rats to environmental tobacco smoke during cerebellar development alters behavior and perturbs mitochondrial energetics.

Pain-related depression of the mesolimbic dopamine system in rats: expression, blockade by analgesics, and role of endogenous κ-opioids.

Rat nucleus accumbens core astrocytes modulate reward and the motivation to self-administer ethanol after abstinence.

Differential roles of α6β2* and α4β2* neuronal nicotinic receptors in nicotine- and cocaine-conditioned reward in mice.

Interactive HIV-1 Tat and morphine-induced synaptodendritic injury is triggered through focal disruptions in Na⁺ influx, mitochondrial instability, and Ca²⁺ overload.

Targeting the Small- and Intermediate-Conductance Ca-Activated Potassium Channels: The Drug-Binding Pocket at the Channel/Calmodulin Interface.

Differential response of glial fibrillary acidic protein-positive astrocytes in the rat prefrontal cortex following ethanol self-administration.