Jeffrey A. Kleim

Motor map expansion following repeated cortical and limbic seizures is related to synaptic potentiation.

Motor learning-dependent synaptogenesis is localized to functionally reorganized motor cortex.

Exercise induces angiogenesis but does not alter movement representations within rat motor cortex.

Synapse formation is associated with memory storage in the cerebellum.

Learning-induced multiple synapse formation in rat cerebellar cortex.

Long-Evans rats have a larger cortical topographic representation of movement than Fischer-344 rats: a microstimulation study of motor cortex in naïve and skilled reaching-trained rats.

A brain adaptation view of plasticity: is synaptic plasticity an overly limited concept?

Functional organization of adult motor cortex is dependent upon continued protein synthesis.

Long-Evans and Sprague-Dawley rats have similar skilled reaching success and limb representations in motor cortex but different movements: some cautionary insights into the selection of rat strains for neurobiological motor research.

Motor enrichment and the induction of plasticity before or after brain injury.

Motor cortex stimulation enhances motor recovery and reduces peri-infarct dysfunction following ischemic insult.

Cortical synaptogenesis and motor map reorganization occur during late, but not early, phase of motor skill learning.

Long-term potentiation induces expanded movement representations and dendritic hypertrophy in layer V of rat sensorimotor neocortex.

Preserved ipsilateral-to-lesion motor map organization in the unilateral 6-OHDA-treated rat model of Parkinson's disease.

Evidence for bilateral control of skilled movements: ipsilateral skilled forelimb reaching deficits and functional recovery in rats follow motor cortex and lateral frontal cortex lesions.

Limits on recovery in the corticospinal tract of the rat: partial lesions impair skilled reaching and the topographic representation of the forelimb in motor cortex.

In search of the motor engram: motor map plasticity as a mechanism for encoding motor experience.

A quantitative comparison of synaptic density following perfusion versus immersion fixation in the rat cerebral cortex.

Induction of neocortical long-term depression results in smaller movement representations, fewer excitatory perforated synapses, and more inhibitory synapses.

III STEP: a basic scientist's perspective.

BDNF val66met polymorphism is associated with modified experience-dependent plasticity in human motor cortex.

FGF-2-induced cell proliferation stimulates anatomical, neurophysiological and functional recovery from neonatal motor cortex injury.

Motor training induces experience-specific patterns of plasticity across motor cortex and spinal cord.

Motor learning induces astrocytic hypertrophy in the cerebellar cortex.

Systematic assessment of training-induced changes in corticospinal output to hand using frameless stereotaxic transcranial magnetic stimulation.

Rat models of upper extremity impairment in stroke.

A novel phosphodiesterase type 4 inhibitor, HT-0712, enhances rehabilitation-dependent motor recovery and cortical reorganization after focal cortical ischemia.

Principles of experience-dependent neural plasticity: implications for rehabilitation after brain damage.

电机的"恢复"和"赔偿"意味着什么脑卒中患者？

Brain plasticity and genetic factors.

BDNF val66met polymorphism influences motor system function in the human brain.

Motor cortex reorganization across the lifespan.

The organization of the forelimb representation of the C57BL/6 mouse motor cortex as defined by intracortical microstimulation and cytoarchitecture.

Distributed versus focal cortical stimulation to enhance motor function and motor map plasticity in a rodent model of ischemia.

Neural plasticity: the biological substrate for neurorehabilitation.

Neural plasticity and neurorehabilitation: teaching the new brain old tricks.

Behavioral and neurophysiological correlates of striatal dopamine depletion: a rodent model of Parkinson's disease.