Kathleen Boris-Lawrie

Destiny of unspliced retroviral RNA: ribosome and/or virion?

Nuclear interactions are necessary for translational enhancement by spleen necrosis virus RU5.

RU5 of Mason-Pfizer monkey virus 5' long terminal repeat enhances cytoplasmic expression of human immunodeficiency virus type 1 gag-pol and nonviral reporter RNA.

Human T-cell lymphotropic virus type 1 p12I enhances interleukin-2 production during T-cell activation.

Primary sequence and secondary structure motifs in spleen necrosis virus RU5 confer translational utilization of unspliced human immunodeficiency virus type 1 reporter RNA.

Analysis of synergy between divergent simple retrovirus posttranscriptional control elements.

Long-term infection with retroviral structural gene vector provides protection against bovine leukemia virus disease in rabbits.

Human T lymphotropic virus type-1 p30II alters cellular gene expression to selectively enhance signaling pathways that activate T lymphocytes.

Human T lymphotropic virus type 1 accessory protein p12I modulates calcium-mediated cellular gene expression and enhances p300 expression in T lymphocytes.

Human T-cell leukemia virus open reading frame II encodes a posttranscriptional repressor that is recruited at the level of transcription.

Coordinate enhancement of transgene transcription and translation in a lentiviral vector.

Retrovirus translation initiation: Issues and hypotheses derived from study of HIV-1.

RNA helicase A is necessary for translation of selected messenger RNAs.

Tertiary structural and functional analyses of a viroid RNA motif by isostericity matrix and mutagenesis reveal its essential role in replication.

RNA helicase A interacts with divergent lymphotropic retroviruses and promotes translation of human T-cell leukemia virus type 1.

Bridging fundamental RNA biology, retroviral replication, and oncogenesis: Karen Beemon wins the 2007 Retrovirology Prize.

HIV-1 Tat RNA silencing suppressor activity is conserved across kingdoms and counteracts translational repression of HIV-1.

Mechanisms employed by retroviruses to exploit host factors for translational control of a complicated proteome.

RNA helicase A modulates translation of HIV-1 and infectivity of progeny virions.

Features of double-stranded RNA-binding domains of RNA helicase A are necessary for selective recognition and translation of complex mRNAs.

RNA helicases: emerging roles in viral replication and the host innate response.

Evidence that Lin28 stimulates translation by recruiting RNA helicase A to polysomes.

Tat RNA silencing suppressor activity contributes to perturbation of lymphocyte miRNA by HIV-1.

Thriving under stress: selective translation of HIV-1 structural protein mRNA during Vpr-mediated impairment of eIF4E translation activity.

Determination of host RNA helicases activity in viral replication.

Cellular RNA helicases and HIV-1: insights from genome-wide, proteomic, and molecular studies.

Stress-induced isoforms of MDM2 and MDM4 correlate with high-grade disease and an altered splicing network in pediatric rhabdomyosarcoma.

HIV-1 and two avian retroviral 5' untranslated regions bind orthologous human and chicken RNA binding proteins.

Isolation of Cognate Cellular and Viral Ribonucleoprotein Complexes of HIV-1 RNA Applicable to Proteomic Discovery and Molecular Investigations.

DHX9/RHA Binding to the PBS-Segment of the Genomic RNA during HIV-1 Assembly Bolsters Virion Infectivity.