Sonia Villapol

Antioxidant Cu/Zn SOD: expression in postnatal brain progenitor cells.

Caspase-3 activation in astrocytes following postnatal excitotoxic damage correlates with cytoskeletal remodeling but not with cell death or proliferation.

Bilateral changes after neonatal ischemia in the P7 rat brain.

Distinct spatial and temporal activation of caspase pathways in neurons and glial cells after excitotoxic damage to the immature rat brain.

Substantial migration of SVZ cells to the cortex results in the generation of new neurons in the excitotoxically damaged immature rat brain.

Astrocyte responses after neonatal ischemia: the yin and the yang.

Unilateral blood flow decrease induces bilateral and symmetric responses in the immature brain.

Melatonin promotes myelination by decreasing white matter inflammation after neonatal stroke.

Dual action of NO synthases on blood flow and infarct volume consecutive to neonatal focal cerebral ischemia.

Candesartan, an angiotensin II AT₁-receptor blocker and PPAR-γ agonist, reduces lesion volume and improves motor and memory function after traumatic brain injury in mice.

Inhaled nitric oxide reduces brain damage by collateral recruitment in a neonatal stroke model.

TGF-β superfamily gene expression and induction of the Runx1 transcription factor in adult neurogenic regions after brain injury.

Commercially available angiotensin II At₂ receptor antibodies are nonspecific.

Smad3 deficiency increases cortical and hippocampal neuronal loss following traumatic brain injury.

Temporal patterns of cortical proliferation of glial cell populations after traumatic brain injury in mice.

Temporal dynamics of cerebral blood flow, cortical damage, apoptosis, astrocyte-vasculature interaction and astrogliosis in the pericontusional region after traumatic brain injury.

Neuroprotective effects of angiotensin receptor blockers.

Polypathology and dementia after brain trauma: Does brain injury trigger distinct neurodegenerative diseases, or should they be classified together as traumatic encephalopathy?

Neurorestoration after traumatic brain injury through angiotensin II receptor blockage.

Hepatic expression of serum amyloid A1 is induced by traumatic brain injury and modulated by telmisartan.

Deleting IGF-1 receptor from forebrain neurons confers neuroprotection during stroke and upregulates endocrine somatotropin.

Dendritic Spine Loss and Chronic White Matter Inflammation in a Mouse Model of Highly Repetitive Head Trauma.

Consequences of hepatic damage after traumatic brain injury: current outlook and potential therapeutic targets.

Protease induced plasticity: matrix metalloproteinase-1 promotes neurostructural changes through activation of protease activated receptor 1.

NOX2 deficiency alters macrophage phenotype through an IL-10/STAT3 dependent mechanism: implications for traumatic brain injury.

Reduced cortical excitatory synapse number in APOE4 mice is associated with increased calcineurin activity.

Sexual dimorphism in the inflammatory response to traumatic brain injury.