Arne Heydorn

Inhibition of quorum sensing in Pseudomonas aeruginosa biofilm bacteria by a halogenated furanone compound.

Statistical analysis of Pseudomonas aeruginosa biofilm development: impact of mutations in genes involved in twitching motility, cell-to-cell signaling, and stationary-phase sigma factor expression.

Metabolic commensalism and competition in a two-species microbial consortium.

Biofilm formation by Pseudomonas aeruginosa wild type, flagella and type IV pili mutants.

Distinct in vitro interaction pattern of dopamine receptor subtypes with adaptor proteins involved in post-endocytotic receptor targeting.

Molecular analysis of rugosity in a Vibrio cholerae O1 El Tor phase variant.

Identification of a novel site within G protein alpha subunits important for specificity of receptor-G protein interaction.

A library of 7TM receptor C-terminal tails. Interactions with the proposed post-endocytic sorting proteins ERM-binding phosphoprotein 50 (EBP50), N-ethylmaleimide-sensitive factor (NSF), sorting nexin 1 (SNX1), and G protein-coupled receptor-associated sorting protein (GASP).

Stratified growth in Pseudomonas aeruginosa biofilms.

A highly conserved glycine within linker I and the extreme C terminus of G protein alpha subunits interact cooperatively in switching G protein-coupled receptor-to-effector specificity.

High content screening for G protein-coupled receptors using cell-based protein translocation assays.

Protein translocation assays: key tools for accessing new biological information with high-throughput microscopy.

A simple cell-based HTS assay system to screen for inhibitors of p53-Hdm2 protein-protein interactions.

The angiotensin type 1 receptor activates extracellular signal-regulated kinases 1 and 2 by G protein-dependent and -independent pathways in cardiac myocytes and langendorff-perfused hearts.

Differential extracellular signal-regulated kinases 1 and 2 activation by the angiotensin type 1 receptor supports distinct phenotypes of cardiac myocytes.

A complex multilevel attack on Pseudomonas aeruginosa algT/U expression and AlgT/U activity results in the loss of alginate production.