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Department of Biochemistry and Molecular Biology,
Division of Cardiovascular Diseases
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Cardiac hypertrophy involves both myocyte hypertrophy and hyperplasia in anemic zebrafish.
PloS one , 2009 | Pubmed ID: 19672293
In vivo protein trapping produces a functional expression codex of the vertebrate proteome.
Nature methods Jun, 2011 | Pubmed ID: 21552255
Haploinsufficiency of target of rapamycin attenuates cardiomyopathies in adult zebrafish.
Circulation research Sep, 2011 | Pubmed ID: 21757652
TOR-autophagy signaling in adult zebrafish models of cardiomyopathy.
Autophagy Jan, 2012 | Pubmed ID: 22186229
Quantifying cardiac functions in embryonic and adult zebrafish.
Methods in molecular biology (Clifton, N.J.) , 2012 | Pubmed ID: 22222517
Target of rapamcyin (TOR)-based therapeutics for cardiomyopathy: Insights from zebrafish genetics.
Cell cycle (Georgetown, Tex.) Feb, 2012 | Pubmed ID: 22262179
Trapping cardiac recessive mutants via expression-based insertional mutagenesis screening.
Circulation research Feb, 2013 | Pubmed ID: 23283723
Cardiac transcriptome and dilated cardiomyopathy genes in zebrafish.
Circulation. Cardiovascular genetics Apr, 2015 | Pubmed ID: 25583992
A modifier screen identifies as a cardiomyopathy susceptibility gene.
JCI insight Sep, 2016 | Pubmed ID: 27642634
Exon- and contraction-dependent functions of titin in sarcomere assembly.
Development (Cambridge, England) Nov, 2016 | Pubmed ID: 27836965
Automated Segmentation of Light-Sheet Fluorescent Imaging to Characterize Experimental Doxorubicin-Induced Cardiac Injury and Repair.
Scientific reports Aug, 2017 | Pubmed ID: 28819303
Mayo Clinic
Xiao Ma*,1,2,3,
Yonghe Ding*,2,3,
Yong Wang2,3,4,
Xiaolei Xu1,2,3
1Clinical and Translational Sciences Track, Mayo Clinic Graduate School of Biomedical Sciences,
2Department of Biochemistry and Molecular Biology, Mayo Clinic,
3Division of Cardiovascular Diseases, Mayo Clinic,
4Institute of Life Science, Beijing University of Chinese Medicine
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