Zsombor Lacza

Hydrogen peroxide acts as an EDHF in the piglet pial vasculature in response to bradykinin.

Activation of mitochondrial ATP-sensitive potassium channels prevents neuronal cell death after ischemia in neonatal rats.

MitoK(ATP) opener, diazoxide, reduces neuronal damage after middle cerebral artery occlusion in the rat.

Mechanisms of pain-induced local cerebral blood flow changes in the rat sensory cortex and thalamus.

Prostacyclin-mediated compensatory mechanism in the coronary circulation during acute NO synthase blockade.

Neural stem cell transplantation in cold lesion: a novel approach for the investigation of brain trauma and repair.

PARP inhibition improves the effectiveness of neural stem cell transplantation in experimental brain trauma.

[Apoptosis in focal brain ischemia].

Heart mitochondria contain functional ATP-dependent K+ channels.

Mitochondrial nitric oxide synthase is not eNOS, nNOS or iNOS.

Investigation of the subunit composition and the pharmacology of the mitochondrial ATP-dependent K+ channel in the brain.

[Neuroprotection in brain ischemia--doubts and hopes].

Lack of mitochondrial nitric oxide production in the mouse brain.

Targeting mitochondrial ATP-sensitive potassium channels--a novel approach to neuroprotection.

Piglet pial arteries respond to N-methyl-D-aspartate in vivo but not in vitro.

Mitochondrial NO and reactive nitrogen species production: does mtNOS exist?

The novel red-fluorescent probe DAR-4M measures reactive nitrogen species rather than NO.

Urotensin-II is a nitric oxide-dependent vasodilator in the pial arteries of the newborn pig.

Graft derived cells with double nuclei in the penumbral region of experimental brain trauma.

Mitochondria produce reactive nitrogen species via an arginine-independent pathway.

Activation of the poly(ADP-ribose) polymerase pathway in human heart failure.

Effects of 7-ketocholesterol on the activity of endothelial poly(ADP-ribose) polymerase and on endothelium-dependent relaxant function.

[Influence of diabetes mellitus on cerebral ischemia and reperfusion injury].

Human heart mitochondria do not produce physiologically relevant quantities of nitric oxide.

Activation of poly(ADP-ribose) polymerase by myocardial ischemia and coronary reperfusion in human circulating leukocytes.

[Astrocytes in health and disease].

Somatosensory pain does not affect total cerebral blood volume.

Mitochondrial nitric oxide synthase: current concepts and controversies.

Intra-mitochondrial poly(ADP-ribosyl)ation: potential role for alpha-ketoglutarate dehydrogenase.

Additive effect of cyclooxygenase and nitric oxide synthase blockade on the cerebrocortical microcirculation.

Mesenchymal stem cells rescue cardiomyoblasts from cell death in an in vitro ischemia model via direct cell-to-cell connections.

Elevated systemic TGF-beta impairs aortic vasomotor function through activation of NADPH oxidase-driven superoxide production and leads to hypertension, myocardial remodeling, and increased plaque formation in apoE(-/-) mice.

Hypersensitivity to thromboxane receptor mediated cerebral vasomotion and CBF oscillations during acute NO-deficiency in rats.

Integration of neuronally predifferentiated human dental pulp stem cells into rat brain in vivo.

Current Developments in the Therapeutic Potential of S-Nitrosoglutathione, an Endogenous NO-Donor Molecule.

Human internal thoracic artery grafts exhibit severe morphological and functional damage and spasmic vasomotion due to oxidative stress.

In vivo evaluation of an injectable premixed radiopaque calcium phosphate cement.

GABAergic signaling in primary lens epithelial and lentoid cells and its involvement in intracellular Ca2+ modulation.

Combined exercise and insulin-like growth factor-1 supplementation induces neurogenesis in old rats, but do not attenuate age-associated DNA damage.

The role of mitochondria in direct cell-to-cell connection dependent rescue of postischemic cardiomyoblasts.

Increased stability of S-nitrosothiol solutions via pH modulations.