Habibeh Khoshbouei

Behavioral reactivity to stress: amplification of stress-induced noradrenergic activation elicits a galanin-mediated anxiolytic effect in central amygdala.

Modulatory effects of galanin in the lateral bed nucleus of the stria terminalis on behavioral and neuroendocrine responses to acute stress.

Modulatory effects of norepinephrine, acting on alpha 1 receptors in the central nucleus of the amygdala, on behavioral and neuroendocrine responses to acute immobilization stress.

Amphetamine-induced dopamine efflux. A voltage-sensitive and intracellular Na+-dependent mechanism.

Interactions of norepinephrine and galanin in the central amygdala and lateral bed nucleus of the stria terminalis modulate the behavioral response to acute stress.

N-terminal phosphorylation of the dopamine transporter is required for amphetamine-induced efflux.

Intracellular Ca2+ regulates amphetamine-induced dopamine efflux and currents mediated by the human dopamine transporter.

Amphetamine induces dopamine efflux through a dopamine transporter channel.

Calmodulin kinase II interacts with the dopamine transporter C terminus to regulate amphetamine-induced reverse transport.

PI3K signaling supports amphetamine-induced dopamine efflux.

Prenatal exposure to benzo(a)pyrene impairs later-life cortical neuronal function.

Amphetamine and methamphetamine differentially affect dopamine transporters in vitro and in vivo.

Methamphetamine reduces LTP and increases baseline synaptic transmission in the CA1 region of mouse hippocampus.

PAH particles perturb prenatal processes and phenotypes: protection from deficits in object discrimination afforded by dampening of brain oxidoreductase following in utero exposure to inhaled benzo(a)pyrene.

α-Synuclein stimulates a dopamine transporter-dependent chloride current and modulates the activity of the transporter.