Andrew E. Sama

Methicillin-resistant Staphylococcus aureus and vancomycin-resistant enterococci: prevalence and predictors of colonization in patients presenting to the emergency department from nursing homes.

IFN-gamma induces high mobility group box 1 protein release partly through a TNF-dependent mechanism.

Extracellular HMGB1 as a proinflammatory cytokine.

Bench to bedside: HMGB1-a novel proinflammatory cytokine and potential therapeutic target for septic patients in the emergency department.

Pathogenic role of HMGB1 in SARS?

Bacterial endotoxin stimulates macrophages to release HMGB1 partly through CD14- and TNF-dependent mechanisms.

Bilateral nursemaid's elbow.

Age-associated time delays in the treatment of acute myocardial infarction with primary percutaneous transluminal coronary angioplasty.

Suppression of HMGB1 release by stearoyl lysophosphatidylcholine:an additional mechanism for its therapeutic effects in experimental sepsis.

The aqueous extract of a popular herbal nutrient supplement, Angelica sinensis, protects mice against lethal endotoxemia and sepsis.

Role of HMGB1 in cardiovascular diseases.

Potential role of high mobility group box 1 in viral infectious diseases.

Elevated high-mobility group box 1 levels in patients with cerebral and myocardial ischemia.

A cardiovascular drug rescues mice from lethal sepsis by selectively attenuating a late-acting proinflammatory mediator, high mobility group box 1.

HMGB1 as a potential therapeutic target.

Cholinergic anti-inflammatory pathway activity and High Mobility Group Box-1 (HMGB1) serum levels in patients with rheumatoid arthritis.

A major ingredient of green tea rescues mice from lethal sepsis partly by inhibiting HMGB1.

Hyperglycemia aggravates endotoxin-induced high mobility group box 1 protein release: yet another reason not to be too sweet.

Therapeutic potential of HMGB1-targeting agents in sepsis.

Caging a Beast in the Inflammation Arena: Use of Chinese Medicinal Herbs to Inhibit a Late Mediator of Lethal Sepsis, HMGB1.

Novel HMGB1-inhibiting therapeutic agents for experimental sepsis.

Spermine protects mice against lethal sepsis partly by attenuating surrogate inflammatory markers.

High mobility group box 1 protein as a potential drug target for infection- and injury-elicited inflammation.

Peripheral administration of fetuin-A attenuates early cerebral ischemic injury in rats.

Immediate cardiac arrest and subsequent development of cardiogenic shock caused by lightning strike.

A hepatic protein, fetuin-A, occupies a protective role in lethal systemic inflammation.

EGCG stimulates autophagy and reduces cytoplasmic HMGB1 levels in endotoxin-stimulated macrophages.

Anti-Inflammatory Role of Fetuin-A in Injury and Infection.