Eugene D. Ponomarev

Translocation of cytoplasmic HSP70 onto the surface of EL-4 cells during apoptosis.

Gamma delta T cell regulation of IFN-gamma production by central nervous system-infiltrating encephalitogenic T cells: correlation with recovery from experimental autoimmune encephalomyelitis.

Gamma delta T cells regulate the extent and duration of inflammation in the central nervous system by a Fas ligand-dependent mechanism.

Development of a culture system that supports adult microglial cell proliferation and maintenance in the resting state.

Microglial cell activation and proliferation precedes the onset of CNS autoimmunity.

Modulation of the cannabinoid CB2 receptor in microglial cells in response to inflammatory stimuli.

CD40 expression by microglial cells is required for their completion of a two-step activation process during central nervous system autoimmune inflammation.

GM-CSF production by autoreactive T cells is required for the activation of microglial cells and the onset of experimental autoimmune encephalomyelitis.

Direct suppression of CNS autoimmune inflammation via the cannabinoid receptor CB1 on neurons and CB2 on autoreactive T cells.

CNS-derived interleukin-4 is essential for the regulation of autoimmune inflammation and induces a state of alternative activation in microglial cells.

IL-13 induces the expression of the alternative activation marker Ym1 in a subset of testicular macrophages.