Department of Surgery
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Over-expression of the transcription factor, ZBP-89, leads to enhancement of the C2C12 myogenic program.
Biochimica et biophysica acta Jul, 2009 | Pubmed ID: 19232372
Cooperative binding of KLF4, pELK-1, and HDAC2 to a G/C repressor element in the SM22α promoter mediates transcriptional silencing during SMC phenotypic switching in vivo.
Circulation research Aug, 2012 | Pubmed ID: 22811558
KLF4 regulates abdominal aortic aneurysm morphology and deletion attenuates aneurysm formation.
Circulation Sep, 2013 | Pubmed ID: 24030402
Inhibition of interleukin-1β decreases aneurysm formation and progression in a novel model of thoracic aortic aneurysms.
Circulation Sep, 2014 | Pubmed ID: 25200056
5-Lipoxygenase pathway in experimental abdominal aortic aneurysms.
Arteriosclerosis, thrombosis, and vascular biology Dec, 2014 | Pubmed ID: 25324573
Attenuation of aortic aneurysms with stem cells from different genders.
The Journal of surgical research Nov, 2015 | Pubmed ID: 25958166
Pharmacologic blockade and genetic deletion of androgen receptor attenuates aortic aneurysm formation.
Journal of vascular surgery 06, 2016 | Pubmed ID: 26817611
ZFP148 (Zinc-Finger Protein 148) Binds Cooperatively With NF-1 (Neurofibromin 1) to Inhibit Smooth Muscle Marker Gene Expression During Abdominal Aortic Aneurysm Formation.
Arteriosclerosis, thrombosis, and vascular biology Jan, 2019 | Pubmed ID: 30580567
Klf4, Klf2, and Zfp148 activate autophagy-related genes in smooth muscle cells during aortic aneurysm formation.
Physiological reports Apr, 2019 | Pubmed ID: 31025534
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