D. Joseph Jerry

Epithelial cell cycling predicts p53 responsiveness to gamma-irradiation during post-natal mammary gland development.

Hormonal control of p53 and chemoprevention.

Knockout and transgenic mice of Trp53: what have we learned about p53 in breast cancer?

BALB/c alleles for Prkdc and Cdkn2a interact to modify tumor susceptibility in Trp53+/- mice.

Loss of heterozygosity occurs via mitotic recombination in Trp53+/- mice and associates with mammary tumor susceptibility of the BALB/c strain.

Estrogen and progesterone regulate radiation-induced p53 activity in mammary epithelium through TGF-beta-dependent pathways.

Sensitivity to DNA damage is a common component of hormone-based strategies for protection of the mammary gland.

Roles for estrogen and progesterone in breast cancer prevention.

Genetic mapping in mice identifies DMBT1 as a candidate modifier of mammary tumors and breast cancer risk.

Mammary tumor modifiers in BALB/cJ mice heterozygous for p53.

Estrogen and progesterone induce persistent increases in p53-dependent apoptosis and suppress mammary tumors in BALB/c-Trp53+/- mice.

Transcriptional responses to estrogen and progesterone in mammary gland identify networks regulating p53 activity.

Regulation of cancer stem cells by p53.

Protein-passivated Fe(3)O(4) nanoparticles: low toxicity and rapid heating for thermal therapy.

Detection and differentiation of normal, cancerous, and metastatic cells using nanoparticle-polymer sensor arrays.

Virus-inspired approach to nonviral gene delivery vehicles.

Identification of a DMBT1 polymorphism associated with increased breast cancer risk and decreased promoter activity.

Activation of host wound responses in breast cancer microenvironment.

Array-based sensing of normal, cancerous, and metastatic cells using conjugated fluorescent polymers.

Pathways contributing to development of spontaneous mammary tumors in BALB/c-Trp53+/- mice.

Estrogens, regulation of p53 and breast cancer risk: a balancing act.

Role of JNK in a Trp53-dependent mouse model of breast cancer.

Repression of Mammary Stem/Progenitor Cells by P53 is Mediated by Notch and Separable from Apoptotic Activity.

Repression of mammary stem/progenitor cells by p53 is mediated by Notch and separable from apoptotic activity.

Map making in the 21st century: charting breast cancer susceptibility pathways in rodent models.

The role of activin in mammary gland development and oncogenesis.

Mitochondrial dysfunction impairs tumor suppressor p53 expression/function.

Radiation acts on the microenvironment to affect breast carcinogenesis by distinct mechanisms that decrease cancer latency and affect tumor type.