Sandra M. Newton

The stress-responsive chaperone alpha-crystallin 2 is required for pathogenesis of Mycobacterium tuberculosis.

Infection biology of a novel alpha-crystallin of Mycobacterium tuberculosis: Acr2.

Effect of deletion or overexpression of the 19-kilodalton lipoprotein Rv3763 on the innate response to Mycobacterium tuberculosis.

Effect of treatment of latent tuberculosis infection on the T cell response to Mycobacterium tuberculosis antigens.

Transmission of Mycobacterium tuberculosis undetected by tuberculin skin testing.

A deletion defining a common Asian lineage of Mycobacterium tuberculosis associates with immune subversion.

IFN-gamma- and TNF-independent vitamin D-inducible human suppression of mycobacteria: the role of cathelicidin LL-37.

Reduction of chemokine secretion in response to mycobacteria in infliximab-treated patients.

Paediatric tuberculosis.

Genetic determination of the effect of post-translational modification on the innate immune response to the 19 kDa lipoprotein of Mycobacterium tuberculosis.

Association between Gc genotype and susceptibility to TB is dependent on vitamin D status.

Azithromycin blocks autophagy and may predispose cystic fibrosis patients to mycobacterial infection.

Functional drug screening reveals anticonvulsants as enhancers of mTOR-independent autophagic killing of Mycobacterium tuberculosis through inositol depletion.

Understanding immune protection against tuberculosis using RNA expression profiling.

Unnatural amino acid analogues of membrane-active helical peptides with anti-mycobacterial activity and improved stability.

Disruption of drug-resistant biofilms using de novo designed short α-helical antimicrobial peptides with idealized facial amphiphilicity.

Innate activation of human primary epithelial cells broadens the host response to Mycobacterium tuberculosis in the airways.

Childhood tuberculosis is associated with decreased abundance of T cell gene transcripts and impaired T cell function.

Mycobacterium tuberculosis Exploits a Molecular Off Switch of the Immune System for Intracellular Survival.

Galleria mellonella - a novel infection model for the Mycobacterium tuberculosis complex.