Kevin J. Woollard

Effects of oral vitamin C on monocyte: endothelial cell adhesion in healthy subjects.

The anti-inflammatory actions of methotrexate are critically dependent upon the production of reactive oxygen species.

Vitamin C supplementation in normal subjects reduces constitutive ICAM-1 expression.

Soluble bio-markers in vascular disease: much more than gauges of disease?

Gravity spun polycaprolactone fibers for applications in vascular tissue engineering: proliferation and function of human vascular endothelial cells.

Alpha-tocopherol supplementation does not affect monocyte endothelial adhesion or C-reactive protein levels but reduces soluble vascular adhesion molecule-1 in the plasma of healthy subjects.

Therapeutic targeting of p-selectin in atherosclerosis.

Conversion of platelets from a proaggregatory to a proinflammatory adhesive phenotype: role of PAF in spatially regulating neutrophil adhesion and spreading.

High-density lipoprotein reduces the human monocyte inflammatory response.

Pathophysiological levels of soluble P-selectin mediate adhesion of leukocytes to the endothelium through Mac-1 activation.

Erythrocyte hemolysis and hemoglobin oxidation promote ferric chloride-induced vascular injury.

Dissociation of pentameric to monomeric C-reactive protein on activated platelets localizes inflammation to atherosclerotic plaques.

High-density lipoprotein: a potent inhibitor of inflammation.

Langerhans cell (LC) proliferation mediates neonatal development, homeostasis, and inflammation-associated expansion of the epidermal LC network.

Monocytes in atherosclerosis: subsets and functions.

A proteomic analysis of C-reactive protein stimulated THP-1 monocytes.

P-selectin antagonism in inflammatory disease.

Neutrophil activation is attenuated by high-density lipoprotein and apolipoprotein A-I in in vitro and in vivo models of inflammation.