Poh-Yi Gan

Th17 cells promote autoimmune anti-myeloperoxidase glomerulonephritis.

Intrinsic renal cell and leukocyte-derived TLR4 aggravate experimental anti-MPO glomerulonephritis.

Toll-like receptor 9 enhances nephritogenic immunity and glomerular leukocyte recruitment, exacerbating experimental crescentic glomerulonephritis.

The Th17-defining transcription factor RORγt promotes glomerulonephritis.

Toll-like receptor 2 induces Th17 myeloperoxidase autoimmunity while Toll-like receptor 9 drives Th1 autoimmunity in murine vasculitis.

Interleukin-17A promotes early but attenuates established disease in crescentic glomerulonephritis in mice.

Mast cells mediate acute kidney injury through the production of TNF.

Mast cell activation and degranulation promotes renal fibrosis in experimental unilateral ureteric obstruction.

Mast cells contribute to peripheral tolerance and attenuate autoimmune vasculitis.

Myeloperoxidase (MPO)-specific CD4+ T cells contribute to MPO-anti-neutrophil cytoplasmic antibody (ANCA) associated glomerulonephritis.

Absence of the lysosomal protein Limp-2 attenuates renal injury in crescentic glomerulonephritis.

FcγRIIB regulates T-cell autoreactivity, ANCA production, and neutrophil activation to suppress anti-myeloperoxidase glomerulonephritis.

T cell mediated autoimmune glomerular disease in mice.

Mouse models of anti-neutrophil cytoplasmic antibody-associated vasculitis.

Cytokines: Names and Numbers You Should Care About.

Myeloperoxidase Peptide-Based Nasal Tolerance in Experimental ANCA-Associated GN.

Renal participation of myeloperoxidase in antineutrophil cytoplasmic antibody (ANCA)-associated glomerulonephritis.

Local IL-17 Production Exerts a Protective Role in Murine Experimental Glomerulonephritis.

Mast Cell Stabilization Ameliorates Autoimmune Anti-Myeloperoxidase Glomerulonephritis.

Biologics for the treatment of autoimmune renal diseases.

T cell receptor assessment in autoimmune disease requires access to the most adjacent immunologically active organ.

Pathogenic Role for γδ T Cells in Autoimmune Anti-Myeloperoxidase Glomerulonephritis.

C5a receptor 1 promotes autoimmunity, neutrophil dysfunction and injury in experimental anti-myeloperoxidase glomerulonephritis.

The C3aR promotes macrophage infiltration and regulates ANCA production but does not affect glomerular injury in experimental anti-myeloperoxidase glomerulonephritis.

Seroresponses and safety of 13-valent pneumococcal conjugate vaccination in kidney transplant recipients.

Natural killer cell function predicts severe infection in kidney transplant recipients.

A simple score can identify kidney transplant recipients at high risk of severe infection over the following 2 years.

Apoptotic Cell-Induced, Antigen-Specific Immunoregulation to Treat Experimental Antimyeloperoxidase GN.

Biologicals targeting T helper cell subset differentiating cytokines are effective in the treatment of murine anti-myeloperoxidase glomerulonephritis.

Tolerogenic Dendritic Cells Attenuate Experimental Autoimmune Antimyeloperoxidase Glomerulonephritis.