Toxicology,
Risk Assessment and Research Division,
Risk Assessment,
and Research Division,
Toxicology, Risk Assessment and Research Division
Dr. Aniagu works as a Toxicologist in the Texas Commission on Environmental Quality Toxicology, Risk Assessment, and Research Division. Dr. Aniagu helps with the derivation of toxicity factors such as Effects Screening Levels (ESLs) in addition to conducting the review of air permit applications in accordance with Agency guidance and protocols. He also helps to conduct advanced toxicological evaluations and risk assessments to determine potential health risks associated with exposure to environmental contaminants, including health effects reviews of ambient air monitoring data. A major part of his work also involves reviewing and coordinating risk assessments for criteria air pollutants (i.e. NAAQS).
Prior Experience and Education
Dr. Stanley Aniagu holds a Bachelor of Pharmacy degree from the Obafemi Awolowo University, Ile-Ife, Nigeria. He also obtained a Master of Science degree in Toxicology as well as a Ph.D. in Environmental & Molecular Toxicology from the University of Birmingham, England. His graduate research focused mainly on non-genotoxic carcinogenesis by environmental chemicals, especially with respect to brominated flame retardants and estrogenic chemicals, which are implicated in cancer, but which also do not cause direct damage to the DNA material.
Dr. Aniagu is a registered Pharmacist with the Boards of Pharmacy in both Texas and Tennessee and regularly participates in the Texas Toxic Substances Coordinating Committee meetings being hosted quarterly at the State Department of Health and Human Services, Austin, TX. In addition, he is a current member of the Society of Toxicology (SOT), the SOT Lone Star Regional Chapter, as well as the SOT Risk Assessment, Carcinogenesis, Inhalation and Occupation and Public Health Specialty Sections. He has over 20 years of combined experience in mechanistic and regulatory toxicology as well as healthcare research and clinical practice.
Aryl hydrocarbon receptor mediates the cardiac developmental toxicity of EOM from PM in P19 embryonic carcinoma cells.
Chemosphere Feb, 2019 | Pubmed ID: 30384306
PM2.5-induced extensive DNA methylation changes in the heart of zebrafish embryos and the protective effect of folic acid.
Environmental pollution (Barking, Essex : 1987) Dec, 2019 | Pubmed ID: 31614245
AHR-mediated oxidative stress contributes to the cardiac developmental toxicity of trichloroethylene in zebrafish embryos.
Journal of hazardous materials 03, 2020 | Pubmed ID: 31699484
AHR-mediated ROS production contributes to the cardiac developmental toxicity of PM2.5 in zebrafish embryos.
The Science of the total environment Jun, 2020 | Pubmed ID: 31837856
Effects of Trichloroethylene on the Expression of Long Intergenic Noncoding RNAs in B6C3F1 Mouse Liver.
Chemical research in toxicology 06, 2020 | Pubmed ID: 31942800
Resveratrol protects against PM2.5-induced heart defects in zebrafish embryos as an antioxidant rather than as an AHR antagonist.
Toxicology and applied pharmacology 07, 2020 | Pubmed ID: 32376357
Protective effects of resveratrol against the cardiac developmental toxicity of trichloroethylene in zebrafish embryos.
Toxicology Jan, 2021 | Pubmed ID: 33524428
AHR/ROS-mediated mitochondria apoptosis contributes to benzo[a]pyrene-induced heart defects and the protective effects of resveratrol.
Toxicology Oct, 2021 | Pubmed ID: 34597721
Cx43 overexpression is involved in the hyper-proliferation effect of trichloroethylene on human embryonic stem cells.
Toxicology Jan, 2022 | Pubmed ID: 34896440
Paternal acrylamide exposure induces transgenerational effects on sperm parameters and learning capability in mice.
Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association Mar, 2022 | Pubmed ID: 35032568
Synergistic protective effects of folic acid and resveratrol against fine particulate matter-induced heart malformations in zebrafish embryos.
Ecotoxicology and environmental safety Aug, 2022 | Pubmed ID: 36068752
PM2.5 induces cardiac malformations via PI3K/akt2/mTORC1 signaling pathway in zebrafish larvae.
Environmental pollution (Barking, Essex : 1987) Feb, 2023 | Pubmed ID: 36804889
PM2.5 induces mitochondrial dysfunction via AHR-mediated cyp1a1 overexpression during zebrafish heart development.
Toxicology Feb, 2023 | Pubmed ID: 36841371
Regulation of Cx43 and its role in trichloroethylene-induced cardiac toxicity in H9C2 rat cardiomyocytes.
Chemosphere Feb, 2023 | Pubmed ID: 36842555
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