Mila Jankovic

The cysteine-rich domain of the macrophage mannose receptor is a multispecific lectin that recognizes chondroitin sulfates A and B and sulfated oligosaccharides of blood group Lewis(a) and Lewis(x) types in addition to the sulfated N-glycans of lutropin.

OcaB regulates transitional B cell selection.

RAGs and regulation of autoantibodies.

AID is required for c-myc/IgH chromosome translocations in vivo.

Role of genomic instability and p53 in AID-induced c-myc-Igh translocations.

Antigen receptor diversification and chromosome translocations.

A role for AID in chromosome translocations between c-myc and the IgH variable region.

MicroRNA-155 suppresses activation-induced cytidine deaminase-mediated Myc-Igh translocation.

Essential role for DNA-PKcs in DNA double-strand break repair and apoptosis in ATM-deficient lymphocytes.

Role of the translocation partner in protection against AID-dependent chromosomal translocations.

Alternative end-joining catalyzes robust IgH locus deletions and translocations in the combined absence of ligase 4 and Ku70.

Alternative end-joining catalyzes class switch recombination in the absence of both Ku70 and DNA ligase 4.

Class switching and meiotic defects in mice lacking the E3 ubiquitin ligase RNF8.

PTIP promotes chromatin changes critical for immunoglobulin class switch recombination.

Human anti-HIV-neutralizing antibodies frequently target a conserved epitope essential for viral fitness.

Activation-induced cytidine deaminase targets DNA at sites of RNA polymerase II stalling by interaction with Spt5.

Translocation-capture sequencing reveals the extent and nature of chromosomal rearrangements in B lymphocytes.

Translocation capture sequencing: a method for high throughput mapping of chromosomal rearrangements.

Robust chromosomal DNA repair via alternative end-joining in the absence of X-ray repair cross-complementing protein 1 (XRCC1).

DNA damage defines sites of recurrent chromosomal translocations in B lymphocytes.

Rif1 prevents resection of DNA breaks and promotes immunoglobulin class switching.

T follicular helper cell dynamics in germinal centers.

Identification of chromosomal translocation hotspots via scan statistics.

B cell super-enhancers and regulatory clusters recruit AID tumorigenic activity.

Epigenetic targeting of activation-induced cytidine deaminase.

HIV-1 integration landscape during latent and active infection.

Plasmodium Infection Promotes Genomic Instability and AID-Dependent B Cell Lymphoma.

Paired quantitative and qualitative assessment of the replication-competent HIV-1 reservoir and comparison with integrated proviral DNA.

The cell cycle restricts activation-induced cytidine deaminase activity to early G1.

Clonal CD4 T cells in the HIV-1 latent reservoir display a distinct gene profile upon reactivation.

Relationship between latent and rebound viruses in a clinical trial of anti-HIV-1 antibody 3BNC117.

Relationship between intact HIV-1 proviruses in circulating CD4 T cells and rebound viruses emerging during treatment interruption.

HIV-specific humoral immune responses by CRISPR/Cas9-edited B cells.

Combination of quadruplex qPCR and next-generation sequencing for qualitative and quantitative analysis of the HIV-1 latent reservoir.

Isolation of single HIV-1 Envelope specific B cells and antibody cloning from immunized rhesus macaques.

Combination anti-HIV-1 antibody therapy is associated with increased virus-specific T cell immunity.

Antigen-responsive CD4+ T cell clones contribute to the HIV-1 latent reservoir.

A Combination of Human Broadly Neutralizing Antibodies against Hepatitis B Virus HBsAg with Distinct Epitopes Suppresses Escape Mutations.

Evolution of antibody immunity to SARS-CoV-2.

Naturally enhanced neutralizing breadth against SARS-CoV-2 one year after infection.

Anti-SARS-CoV-2 receptor-binding domain antibody evolution after mRNA vaccination.

Integration features of intact latent HIV-1 in CD4+ T cell clones contribute to viral persistence.

Increased memory B cell potency and breadth after a SARS-CoV-2 mRNA boost.

Antibody evolution to SARS-CoV-2 after single-dose Ad26.COV2.S vaccine in humans.

Humoral immunity to SARS-CoV-2 elicited by combination COVID-19 vaccination regimens.

Distinct gene expression by expanded clones of quiescent memory CD4 T cells harboring intact latent HIV-1 proviruses.

Memory B cell responses to Omicron subvariants after SARS-CoV-2 mRNA breakthrough infection in humans.