Brian Storrie

Dynamic nucleation of Golgi apparatus assembly from the endoplasmic reticulum in interphase hela cells.

Cisternal rab proteins regulate Golgi apparatus redistribution in response to hypotonic stress.

Maintenance of Golgi apparatus structure in the face of continuous protein recycling to the endoplasmic reticulum: making ends meet.

The steady-state distribution of glycosyltransferases between the Golgi apparatus and the endoplasmic reticulum is approximately 90:10.

Microinjection as a tool to explore small GTPase function.

Capacity of the Golgi apparatus for cargo transport prior to complete assembly.

Both post-Golgi and intra-Golgi cycling affect the distribution of the Golgi phosphoprotein GPP130.

Alternate routes for drug delivery to the cell interior: pathways to the Golgi apparatus and endoplasmic reticulum.

Rab6 regulates both ZW10/RINT-1 and conserved oligomeric Golgi complex-dependent Golgi trafficking and homeostasis.

Bortezomib down-regulates the cell-surface expression of HLA class I and enhances natural killer cell-mediated lysis of myeloma.

Sequestration of mutated alpha1-antitrypsin into inclusion bodies is a cell-protective mechanism to maintain endoplasmic reticulum function.

Using quantitative fluorescence microscopy to probe organelle assembly and membrane trafficking.

Visualizing the von Willebrand factor/glycoprotein Ib-IX axis with a platelet-type von Willebrand disease mutation.

Rab33b and Rab6 are functionally overlapping regulators of Golgi homeostasis and trafficking.

Quantitative immunofluorescence mapping reveals little functional coclustering of proteins within platelet α-granules.

Live-cell assays to identify regulators of ER-to-Golgi trafficking.

Electron tomography reveals Rab6 is essential to the trafficking of trans-Golgi clathrin and COPI-coated vesicles and the maintenance of Golgi cisternal number.

Highly activated and expanded natural killer cells for multiple myeloma immunotherapy.

Are Rab proteins the link between Golgi organization and membrane trafficking?

Rab6a/a' are important Golgi regulators of pro-inflammatory TNF secretion in macrophages.

Rab41 is a novel regulator of Golgi apparatus organization that is needed for ER-to-Golgi trafficking and cell growth.

Distinct sets of Rab6 effectors contribute to ZW10--and COG-dependent Golgi homeostasis.

MCP-1 downregulates MMP-9 export via vesicular redistribution to lysosomes in rat portal fibroblasts.

Does super-resolution fluorescence microscopy obsolete previous microscopic approaches to protein co-localization?

How Rab proteins determine Golgi structure.

Defective platelet autocrine signaling in HPS.

Autophagy is induced upon platelet activation and is essential for hemostasis and thrombosis.

STEM tomography reveals that the canalicular system and α-granules remain separate compartments during early secretion stages in blood platelets.

CINCH (confocal incoherent correlation holography) super resolution fluorescence microscopy based upon FINCH (Fresnel incoherent correlation holography).

Identification of Rab41/6d Effectors Provides an Explanation for the Differential Effects of Rab41/6d and Rab6a/a' on Golgi Organization.

A tip of the cap to procoagulant platelets.

Golgi proteins in circulating human platelets are distributed across non-stacked, scattered structures.

The cellular basis of platelet secretion: Emerging structure/function relationships.

Editorial: Platelet Secretion.

Managing the Introduction of Super-Resolution Microscopy into a Core Facility.

Cellubrevin/vesicle-associated membrane protein-3-mediated endocytosis and trafficking regulate platelet functions.

Rab proteins as major determinants of the Golgi complex structure.

GADD34 Function in Protein Trafficking Promotes Adaptation to Hyperosmotic Stress in Human Corneal Cells.

Alterations in platelet secretion differentially affect thrombosis and hemostasis.

SNARE-dependent membrane fusion initiates α-granule matrix decondensation in mouse platelets.

Autophagy in Platelets.

Epistatic Analysis of the Contribution of Rabs and Kifs to CATCHR Family Dependent Golgi Organization.

3D ultrastructural analysis of α-granule, dense granule, mitochondria, and canalicular system arrangement in resting human platelets.

Canalicular system reorganization during mouse platelet activation as revealed by 3D ultrastructural analysis.

Structural analysis of resting mouse platelets by 3D-EM reveals an unexpected variation in α-granule shape.

Rab6 is required for rapid, cisternal-specific, intra-Golgi cargo transport.

Platelet α-granule cargo packaging and release are affected by the luminal proteoglycan, serglycin.

Dense cellular segmentation for EM using 2D-3D neural network ensembles.

Venous puncture wound hemostasis results in a vaulted thrombus structured by locally nucleated platelet aggregates.

High-Pressure Freezing Followed by Freeze Substitution: An Optimal Electron Microscope Technique to Study Golgi Apparatus Organization and Membrane Trafficking.

Tethered platelet capture provides a mechanism for restricting circulating platelet activation to the wound site.

A missense mouse model for Smith-McCort dysplasia shows bone resorption defects and altered protein glycosylation.

Deep learning, 3D ultrastructural analysis reveals quantitative differences in platelet and organelle packing in COVID-19/SARSCoV2 patient-derived platelets.