Kendal Broadie

Developmental regulation of glutamate receptor field size by nonvesicular glutamate release.

Wrestling with pleiotropy: genomic and topological analysis of the yeast gene expression network.

Integrins regulate DLG/FAS2 via a CaM kinase II-dependent pathway to mediate synapse elaboration and stabilization during postembryonic development.

Response: meaningless minis?

Electrophysiological analysis of synaptic transmission in central neurons of Drosophila larvae.

Living synaptic vesicle marker: synaptotagmin-GFP.

Establishing and sculpting the synapse in Drosophila and C. elegans.

The synaptic vesicle cycle: exocytosis and endocytosis in Drosophila and C. elegans.

Drosophila sec10 is required for hormone secretion but not general exocytosis or neurotransmission.

Cellular bases of behavioral plasticity: establishing and modifying synaptic circuits in the Drosophila genetic system.

Synaptic Drosophila UNC-13 is regulated by antagonistic G-protein pathways via a proteasome-dependent degradation mechanism.

Mutation and activation of Galpha s similarly alters pre- and postsynaptic mechanisms modulating neurotransmission.

The ubiquitin proteasome system acutely regulates presynaptic protein turnover and synaptic efficacy.

Techniques to dissect cellular and subcellular function in the Drosophila nervous system.

Axon pruning: an active role for glial cells.

The Drosophila fragile X-related gene regulates axoneme differentiation during spermatogenesis.

The hereditary spastic paraplegia gene, spastin, regulates microtubule stability to modulate synaptic structure and function.

Cellular bases of activity-dependent paralysis in Drosophila stress-sensitive mutants.

Ceramidase regulates synaptic vesicle exocytosis and trafficking.

Rolling blackout, a newly identified PIP2-DAG pathway lipase required for Drosophila phototransduction.

Synapse scaffolding: intersection of endocytosis and growth.

The Drosophila metabotropic glutamate receptor DmGluRA regulates activity-dependent synaptic facilitation and fine synaptic morphology.

The Drosophila fragile X gene negatively regulates neuronal elaboration and synaptic differentiation.

Protein expression profiling of the drosophila fragile X mutant brain reveals up-regulation of monoamine synthesis.

Fathoming fragile X in fruit flies.

Lipid regulation of the synaptic vesicle cycle.

Translational complexity of the fragile x mental retardation protein: insights from the fly.

An essential Drosophila glutamate receptor subunit that functions in both central neuropil and neuromuscular junction.

Rolling blackout is required for synaptic vesicle exocytosis.

In vivo assay of presynaptic microtubule cytoskeleton dynamics in Drosophila.

The ubiquitin-proteasome system postsynaptically regulates glutamatergic synaptic function.

Proteasome function is required to maintain muscle cellular architecture.

Presynaptic establishment of the synaptic cleft extracellular matrix is required for post-synaptic differentiation.

Drosophila fragile X mental retardation protein and metabotropic glutamate receptor A convergently regulate the synaptic ratio of ionotropic glutamate receptor subclasses.

CNS-derived glia ensheath peripheral nerves and mediate motor root development.

Roles of ubiquitination at the synapse.

Mechanistic relationships between Drosophila fragile X mental retardation protein and metabotropic glutamate receptor A signaling.

Drosophila fragile X mental retardation protein developmentally regulates activity-dependent axon pruning.

Mutational analysis establishes a critical role for the N terminus of fragile X mental retardation protein FMRP.

Presynaptic calcium channel localization and calcium-dependent synaptic vesicle exocytosis regulated by the Fuseless protein.

Temporal requirements of the fragile X mental retardation protein in the regulation of synaptic structure.

Neuronal loss of Drosophila NPC1a causes cholesterol aggregation and age-progressive neurodegeneration.

Excess protein synthesis in Drosophila fragile X mutants impairs long-term memory.

Two peptide transmitters co-packaged in a single neurosecretory vesicle.

Metabotropic glutamate receptor-mediated use-dependent down-regulation of synaptic excitability involves the fragile X mental retardation protein.

Motor deficit in a Drosophila model of mucolipidosis type IV due to defective clearance of apoptotic cells.

Rolling blackout is required for bulk endocytosis in non-neuronal cells and neuronal synapses.

The fragile X mental retardation protein in circadian rhythmicity and memory consolidation.

Presynaptic secretion of mind-the-gap organizes the synaptic extracellular matrix-integrin interface and postsynaptic environments.

Activity-dependent modulation of neural circuit synaptic connectivity.

Temporal requirements of the fragile x mental retardation protein in modulating circadian clock circuit synaptic architecture.

Structural mass spectrometry analysis of lipid changes in a Drosophila epilepsy model brain.

Fragile X mental retardation protein has a unique, evolutionarily conserved neuronal function not shared with FXR1P or FXR2P.

The nonsense-mediated decay pathway maintains synapse architecture and synaptic vesicle cycle efficacy.

The fragile X mental retardation protein developmentally regulates the strength and fidelity of calcium signaling in Drosophila mushroom body neurons.

Anterograde Jelly belly ligand to Alk receptor signaling at developing synapses is regulated by Mind the gap.

Drosophila rolling blackout displays lipase domain-dependent and -independent endocytic functions downstream of dynamin.

The Cdc42-selective GAP rich regulates postsynaptic development and retrograde BMP transsynaptic signaling.

Genetic controls balancing excitatory and inhibitory synaptogenesis in neurodevelopmental disorder models.

Glycosylated synaptomatrix regulation of trans-synaptic signaling.

Fragile X mental retardation protein is required for programmed cell death and clearance of developmentally-transient peptidergic neurons.

Drosophila modeling of heritable neurodevelopmental disorders.

Neural circuit architecture defects in a Drosophila model of Fragile X syndrome are alleviated by minocycline treatment and genetic removal of matrix metalloproteinase.

Extracellular matrix and its receptors in Drosophila neural development.

Molecular and genetic analysis of the Drosophila model of fragile X syndrome.

In vivo neuronal function of the fragile X mental retardation protein is regulated by phosphorylation.

Structure-function analysis of endogenous lectin mind-the-gap in synaptogenesis.

GABAergic circuit dysfunction in the Drosophila Fragile X syndrome model.

Two protein N-acetylgalactosaminyl transferases regulate synaptic plasticity by activity-dependent regulation of integrin signaling.

Overelaborated synaptic architecture and reduced synaptomatrix glycosylation in a Drosophila classic galactosemia disease model.

Activity-dependent FMRP requirements in development of the neural circuitry of learning and memory.

Two classes of matrix metalloproteinases reciprocally regulate synaptogenesis.

A fully automated Drosophila olfactory classical conditioning and testing system for behavioral learning and memory assessment.

Neuron class-specific requirements for Fragile X Mental Retardation Protein in critical period development of calcium signaling in learning and memory circuitry.

Synaptic roles for phosphomannomutase type 2 in a new Drosophila congenital disorder of glycosylation disease model.

Coordinated movement, neuromuscular synaptogenesis and trans-synaptic signaling defects in Drosophila galactosemia models.

Developmental experience-dependent plasticity in the first synapse of the Drosophila olfactory circuit.

Neural Circuits: Reduced Inhibition in Fragile X Syndrome.

Secreted tissue inhibitor of matrix metalloproteinase restricts -synaptic signaling to coordinate synaptogenesis.

Fragile X Mental Retardation Protein Requirements in Activity-Dependent Critical Period Neural Circuit Refinement.

ESCRT-III Membrane Trafficking Misregulation Contributes To Fragile X Syndrome Synaptic Defects.

Multifarious Functions of the Fragile X Mental Retardation Protein.

Notum coordinates synapse development via extracellular regulation of Wingless trans-synaptic signaling.

Fragile X Mental Retardation Protein Restricts Small Dye Iontophoresis Entry into Central Neurons.

Neuronal activity drives FMRP- and HSPG-dependent matrix metalloproteinase function required for rapid synaptogenesis.

Newly Identified Electrically Coupled Neurons Support Development of the Giant Fiber Model Circuit.

Activity-Dependent Remodeling of Olfactory Sensory Neuron Brain Innervation during an Early-Life Critical Period.

Fragile X Mental Retardation Protein positively regulates PKA anchor Rugose and PKA activity to control actin assembly in learning/memory circuitry.

Carrier of Wingless (Cow) Regulation of Neuromuscular Junction Development.

Genetic background mutations drive neural circuit hyperconnectivity in a fragile X syndrome model.

Extracellular heparan sulfate proteoglycans and glycan-binding lectins orchestrate -synaptic signaling.

FMRP-PKA Activity Negative Feedback Regulates RNA Binding-Dependent Fibrillation in Brain Learning and Memory Circuitry.

Neuron-Specific FMRP Roles in Experience-Dependent Remodeling of Olfactory Brain Innervation during an Early-Life Critical Period.

Neuronal fragile X mental retardation protein activates glial insulin receptor mediated PDF-Tri neuron developmental clearance.

Secreted C-type lectin regulation of neuromuscular junction synaptic vesicle dynamics modulates coordinated movement.

Temporally and Spatially Localized PKA Activity within Learning and Memory Circuitry Regulated by Network Feedback.

RNA-binding FMRP and Staufen sequentially regulate the Coracle scaffold to control synaptic glutamate receptor and bouton development.

Dysregulation of BMP, Wnt, and Insulin Signaling in Fragile X Syndrome.

FMRP activity and control of Csw/SHP2 translation regulate MAPK-dependent synaptic transmission.

Fragile X mental retardation protein coordinates neuron-to-glia communication for clearance of developmentally transient brain neurons.

Glycosphingolipids are linked to elevated neurotransmission and neurodegeneration in a Drosophila model of Niemann Pick type C.

Muscle cofilin alters neuromuscular junction postsynaptic development to strengthen functional neurotransmission.

Use-dependent, untapped dual kinase signaling localized in brain learning circuitry.