Kory J. Lavine

Reciprocal epithelial-mesenchymal FGF signaling is required for cecal development.

FGF18 is required for early chondrocyte proliferation, hypertrophy and vascular invasion of the growth plate.

Rebuilding the coronary vasculature: hedgehog as a new candidate for pharmacologic revascularization.

FGF9 regulates early hypertrophic chondrocyte differentiation and skeletal vascularization in the developing stylopod.

FGF9 and SHH regulate mesenchymal Vegfa expression and development of the pulmonary capillary network.

Fibroblast growth factors and Hedgehogs: at the heart of the epicardial signaling center.

The Dynamic Role of Cardiac Macrophages in Aging and Disease.

Novel tool to suppress cell proliferation in vivo demonstrates that myocardial and coronary vascular growth represent distinct developmental programs.

Hedgehog signaling is critical for maintenance of the adult coronary vasculature in mice.

Hedgehog signaling to distinct cell types differentially regulates coronary artery and vein development.

Shared circuitry: developmental signaling cascades regulate both embryonic and adult coronary vasculature.

Rescue of cardiomyopathy in peroxisome proliferator-activated receptor-alpha transgenic mice by deletion of lipoprotein lipase identifies sources of cardiac lipids and peroxisome proliferator-activated receptor-alpha activators.

The development of myocardial fibrosis in transgenic mice with targeted overexpression of tumor necrosis factor requires mast cell-fibroblast interactions.

Coronary collaterals predict improved survival and allograft function in patients with coronary allograft vasculopathy.

Repetitive myocardial ischemia promotes coronary growth in the adult mammalian heart.

Fibroblast growth factor receptor 1 signaling in adult cardiomyocytes increases contractility and results in a hypertrophic cardiomyopathy.

Embryonic and adult-derived resident cardiac macrophages are maintained through distinct mechanisms at steady state and during inflammation.

Origin and functions of tissue macrophages.

Endothelial cell FGF signaling is required for injury response but not for vascular homeostasis.

Rethinking Phase II Clinical Trial Design in Heart Failure.

Distinct macrophage lineages contribute to disparate patterns of cardiac recovery and remodeling in the neonatal and adult heart.

Necrotic myocardial cells release damage-associated molecular patterns that provoke fibroblast activation in vitro and trigger myocardial inflammation and fibrosis in vivo.

Intra-Aortic Balloon Counterpulsation in Patients With Chronic Heart Failure and Cardiogenic Shock: Clinical Response and Predictors of Stabilization.

Stromal-Initiated Changes in the Bone Promote Metastatic Niche Development.

Primitive Embryonic Macrophages are Required for Coronary Development and Maturation.

Proliferation and Recruitment Contribute to Myocardial Macrophage Expansion in Chronic Heart Failure.

Heart-resident CCR2 macrophages promote neutrophil extravasation through TLR9/MyD88/CXCL5 signaling.

Skeletal muscle inflammation and atrophy in heart failure.

Macrophages Facilitate Electrical Conduction in the Heart.

Pediatric and adult dilated cardiomyopathy represent distinct pathological entities.

Cardiac Lymphatic Vessels, Transport, and Healing of the Infarcted Heart.

A CD103 Conventional Dendritic Cell Surveillance System Prevents Development of Overt Heart Failure during Subclinical Viral Myocarditis.

Recognition of self-DNA drives cardiac inflammation: why broken hearts fail.

Visualization of Monocytic Cells in Regressing Atherosclerotic Plaques by Intravital 2-Photon and Positron Emission Tomography-Based Imaging-Brief Report.

Spleen-derived classical monocytes mediate lung ischemia-reperfusion injury through IL-1β.

The human heart contains distinct macrophage subsets with divergent origins and functions.

Slicing Into Human Translational Cardiovascular Biology.

The Macrophage in Cardiac Homeostasis and Disease: JACC Macrophage in CVD Series (Part 4).

Self-renewing resident cardiac macrophages limit adverse remodeling following myocardial infarction.

Tissue Resident CCR2- and CCR2+ Cardiac Macrophages Differentially Orchestrate Monocyte Recruitment and Fate Specification Following Myocardial Injury.

Molecular Imaging Visualizes Recruitment of Inflammatory Monocytes and Macrophages to the Injured Heart.

Publisher Correction: Self-renewing resident cardiac macrophages limit adverse remodeling following myocardial infarction.

Immunomodulatory role of non-neuronal cholinergic signaling in myocardial injury.

Macrophage Plasticity and Function in the Eye and Heart.

New Approaches to Target Inflammation in Heart Failure: Harnessing Insights from Studies of Immune Cell Diversity.

TFEB activation in macrophages attenuates postmyocardial infarction ventricular dysfunction independently of ATG5-mediated autophagy.

Targeted PET Imaging of Chemokine Receptor 2-Positive Monocytes and Macrophages in the Injured Heart.

Chemokine Receptor 2-targeted Molecular Imaging in Pulmonary Fibrosis. A Clinical Trial.

Eosinophils Confer Protection Following Myocardial Infarction.

Heterogeneous origins and functions of mouse skeletal muscle-resident macrophages.

A Minimal-Invasive Approach for Standardized Induction of Myocardial Infarction in Mice.

Beyond genomics-technological advances improving the molecular characterization and precision treatment of heart failure.

Limited proliferation capacity of aortic intima resident macrophages requires monocyte recruitment for atherosclerotic plaque progression.

Lymphatic drainage from bronchus-associated lymphoid tissue in tolerant lung allografts promotes peripheral tolerance.

SARS-CoV-2 Infects Human Engineered Heart Tissues and Models COVID-19 Myocarditis.

SVEP1 is a human coronary artery disease locus that promotes atherosclerosis.

Resident cardiac macrophages mediate adaptive myocardial remodeling.

Selective loss of resident macrophage-derived insulin-like growth factor-1 abolishes adaptive cardiac growth to stress.

Pediatric and adult dilated cardiomyopathy are distinguished by distinct biomarker profiles.

Myocarditis after Covid-19 mRNA Vaccination.

Leveraging FPR2 Agonists to Resolve Inflammation and Improve Outcomes Following Myocardial Infarction.

CCL17 Aggravates Myocardial Injury by Suppressing Recruitment of Regulatory T Cells.

Cell specific peripheral immune responses predict survival in critical COVID-19 patients.

Derivation of extra-embryonic and intra-embryonic macrophage lineages from human pluripotent stem cells.

The dynamic cardiac cellular landscape: visualization by molecular imaging.

Meteorin-like promotes heart repair through endothelial KIT receptor tyrosine kinase.