Jose L. Lopez-Ribot

Molecular mechanisms of fluconazole resistance in Candida dubliniensis isolates from human immunodeficiency virus-infected patients with oropharyngeal candidiasis.

In vitro activity of caspofungin (MK-0991) against Candida albicans clinical isolates displaying different mechanisms of azole resistance.

Investigation of multidrug efflux pumps in relation to fluconazole resistance in Candida albicans biofilms.

Replacement of Candida albicans with C. dubliniensis in human immunodeficiency virus-infected patients with oropharyngeal candidiasis treated with fluconazole.

The filamentation pathway controlled by the Efg1 regulator protein is required for normal biofilm formation and development in Candida albicans.

In vitro activity of caspofungin against Candida albicans biofilms.

In vitro pharmacodynamic properties of three antifungal agents against preformed Candida albicans biofilms determined by time-kill studies.

Inhibition of Candida albicans biofilm formation by farnesol, a quorum-sensing molecule.

Engineered control of cell morphology in vivo reveals distinct roles for yeast and filamentous forms of Candida albicans during infection.

Antifungal combinations against Candida albicans biofilms in vitro.

Changes in susceptibility to posaconazole in clinical isolates of Candida albicans.

The C-terminal antibody binding domain of Candida albicans mp58 represents a protective epitope during candidiasis.

Caspofungin resistance in Candida albicans: correlating clinical outcome with laboratory susceptibility testing of three isogenic isolates serially obtained from a patient with progressive Candida esophagitis.

Inducible defense mechanism against nitric oxide in Candida albicans.

Antibody response to Candida albicans cell wall antigens.

Denture stomatitis: a role for Candida biofilms.

Candida biofilms: an update.

Candida albicans biofilms: more than filamentation.

A role for Efg1p in Candida albicans interactions with extracellular matrices.

Use of genome information for the study of the pathogenesis of fungal infections and the development of diagnostic tools.

Drosophila melanogaster as a facile model for large-scale studies of virulence mechanisms and antifungal drug efficacy in Candida species.

Proteomics for the analysis of the Candida albicans biofilm lifestyle.

Inhibition of filamentation can be used to treat disseminated candidiasis.

Biofilm formation by Candida albicans mutants for genes coding fungal proteins exhibiting the eight-cysteine-containing CFEM domain.

A proteomic-based approach for the identification of Candida albicans protein components present in a subunit vaccine that protects against disseminated candidiasis.

Proteomics to study Candida albicans biology and pathogenicity.

Inhibition on Candida albicans biofilm formation using divalent cation chelators (EDTA).

Use of a genetically engineered strain to evaluate the pathogenic potential of yeast cell and filamentous forms during Candida albicans systemic infection in immunodeficient mice.

UME6, a novel filament-specific regulator of Candida albicans hyphal extension and virulence.

A seed and feed model for the formation of Candida albicans biofilms under flow conditions using an improved modified Robbins device.

Candida albicans-endothelial cell interactions: a key step in the pathogenesis of systemic candidiasis.

A simple and reproducible 96-well plate-based method for the formation of fungal biofilms and its application to antifungal susceptibility testing.

Effect of tunicamycin on Candida albicans biofilm formation and maintenance.

Expression levels of a filament-specific transcriptional regulator are sufficient to determine Candida albicans morphology and virulence.

Efficacy of a genetically engineered Candida albicans tet-NRG1 strain as an experimental live attenuated vaccine against hematogenously disseminated candidiasis.

Design of a simple model of Candida albicans biofilms formed under conditions of flow: development, architecture, and drug resistance.

Adhesion of Candida albicans to endothelial cells under physiological conditions of flow.

Treatment and prevention of Candida albicans biofilms with caspofungin in a novel central venous catheter murine model of candidiasis.

Characteristics of Candida albicans biofilms grown in a synthetic urine medium.

Presence of extracellular DNA in the Candida albicans biofilm matrix and its contribution to biofilms.

Dispersion as an important step in the Candida albicans biofilm developmental cycle.

The transcriptional regulator Nrg1p controls Candida albicans biofilm formation and dispersion.

An easy and economical in vitro method for the formation of Candida albicans biofilms under continuous conditions of flow.

Effects of fluconazole, amphotericin B, and caspofungin on Candida albicans biofilms under conditions of flow and on biofilm dispersion.

Validation of the tetracycline regulatable gene expression system for the study of the pathogenesis of infectious disease.

Addition of DNase improves the in vitro activity of antifungal drugs against Candida albicans biofilms.

Hsp90 governs dispersion and drug resistance of fungal biofilms.

Effect of exogenous administration of Candida albicans autoregulatory alcohols in a murine model of hematogenously disseminated candidiasis.

Sodium hypochlorite, chlorhexidine gluconate, and commercial denture cleansers as disinfecting agents against Candida albicans: An in vitro comparison study.