Mayumi L. Prins

The effects of combined fluid percussion traumatic brain injury and unilateral entorhinal deafferentation on the juvenile rat brain.

Developing experimental models to address traumatic brain injury in children.

Increased cerebral uptake and oxidation of exogenous betaHB improves ATP following traumatic brain injury in adult rats.

Age-dependent reduction of cortical contusion volume by ketones after traumatic brain injury.

Noninvasive measurement of cardiovascular function in mice with high-temporal-resolution small-animal PET.

Is being plastic fantastic? Mechanisms of altered plasticity after developmental traumatic brain injury.

Induction of monocarboxylate transporter 2 expression and ketone transport following traumatic brain injury in juvenile and adult rats.

In vivo quantitation of glucose metabolism in mice using small-animal PET and a microfluidic device.

Cerebral metabolic adaptation and ketone metabolism after brain injury.

Diet, ketones, and neurotrauma.

The effects of age and ketogenic diet on local cerebral metabolic rates of glucose after controlled cortical impact injury in rats.

Hitting a moving target: Basic mechanisms of recovery from acquired developmental brain injury.

Repeat traumatic brain injury in the juvenile rat is associated with increased axonal injury and cognitive impairments.

Molecular and physiological responses to juvenile traumatic brain injury: focus on growth and metabolism.

Repeat traumatic brain injury in the developing brain.

Cerebral ketone metabolism during development and injury.

Repeated mild traumatic brain injury: mechanisms of cerebral vulnerability.

Traumatic brain injury and diet.

Metabolic Response of Pediatric Traumatic Brain Injury.

Ketogenic diet decreases oxidative stress and improves mitochondrial respiratory complex activity.

Influence of Glycemic Control on Endogenous Circulating Ketone Concentrations in Adults Following Traumatic Brain Injury.

It's Not All Fun and Games: Sports, Concussions, and Neuroscience.

Repeat Mild Traumatic Brain Injury in Adolescent Rats Increases Subsequent β-Amyloid Pathogenesis.

Glucose metabolism in pediatric traumatic brain injury.

Concussion: pathophysiology and clinical translation.

Mechanisms underlying vulnerabilities after repeat mild traumatic brain injuries.

Bridging the gap: Mechanisms of plasticity and repair after pediatric TBI.