Nalini Ramarao

The InhA1 metalloprotease allows spores of the B. cereus group to escape macrophages.

FlhA influences Bacillus thuringiensis PlcR-regulated gene transcription, protein production, and virulence.

Adhesion and cytotoxicity of Bacillus cereus and Bacillus thuringiensis to epithelial cells are FlhA and PlcR dependent, respectively.

Capping of actin filaments by vinculin activated by the Shigella IpaA carboxyl-terminal domain.

Growth-related variations in the Bacillus cereus secretome.

The YvfTU two-component system is involved in plcR expression in Bacillus cereus.

Biofilm formation and cell surface properties among pathogenic and nonpathogenic strains of the Bacillus cereus group.

The InhA metalloproteases of Bacillus cereus contribute concomitantly to virulence.

InhA1, NprA, and HlyII as candidates for markers to differentiate pathogenic from nonpathogenic Bacillus cereus strains.

CwpFM (EntFM) is a Bacillus cereus potential cell wall peptidase implicated in adhesion, biofilm formation, and virulence.

Haemolysin II is a Bacillus cereus virulence factor that induces apoptosis of macrophages.

Bacillus anthracis protease InhA increases blood-brain barrier permeability and contributes to cerebral hemorrhages.

Trypan blue dye enters viable cells incubated with the pore-forming toxin HlyII of Bacillus cereus.

Bacillus anthracis protease InhA regulates BslA-mediated adhesion in human endothelial cells.

Necrotrophism is a quorum-sensing-regulated lifestyle in Bacillus thuringiensis.

How the insect pathogen bacteria Bacillus thuringiensis and Xenorhabdus/Photorhabdus occupy their hosts.

Pathogenic potential of B. cereus strains revealed by phenotypic analysis.