Irving C. Allen

Thromboxane A2 induces airway constriction through an M3 muscarinic acetylcholine receptor-dependent mechanism.

Expression and function of NPSR1/GPRA in the lung before and after induction of asthma-like disease.

The NLRP3 inflammasome mediates in vivo innate immunity to influenza A virus through recognition of viral RNA.

Cooperation between mast cells and neurons is essential for antigen-mediated bronchoconstriction.

NLRP3 (NALP3, Cryopyrin) facilitates in vivo caspase-1 activation, necrosis, and HMGB1 release via inflammasome-dependent and -independent pathways.

Staphylococcus aureus alpha-hemolysin activates the NLRP3-inflammasome in human and mouse monocytic cells.

Searching for an improved mouse model of allergic airway disease using dual allergen exposures.

The NLRP3 inflammasome functions as a negative regulator of tumorigenesis during colitis-associated cancer.

Granuloma formation and host defense in chronic Mycobacterium tuberculosis infection requires PYCARD/ASC but not NLRP3 or caspase-1.

Cutting edge: NLRP12 controls dendritic and myeloid cell migration to affect contact hypersensitivity.

The inflammasome component NLRP3 impairs antitumor vaccine by enhancing the accumulation of tumor-associated myeloid-derived suppressor cells.

The NLR adaptor ASC/PYCARD regulates DUSP10, mitogen-activated protein kinase (MAPK), and chemokine induction independent of the inflammasome.

NLRX1 protein attenuates inflammatory responses to infection by interfering with the RIG-I-MAVS and TRAF6-NF-κB signaling pathways.

Allergic inflammation induces a persistent mechanistic switch in thromboxane-mediated airway constriction in the mouse.

A NOD to zebrafish models of inflammatory bowel disease pathogenesis.

Staphylococcus aureus α-hemolysin mediates virulence in a murine model of severe pneumonia through activation of the NLRP3 inflammasome.

Characterization of NLRP12 during the development of allergic airway disease in mice.

TLR2/MyD88/NF-κB pathway, reactive oxygen species, potassium efflux activates NLRP3/ASC inflammasome during respiratory syncytial virus infection.

Analysis of NLRP3 in the development of allergic airway disease in mice.

NLRP12 suppresses colon inflammation and tumorigenesis through the negative regulation of noncanonical NF-κB signaling.

The innate immune sensor NLRC3 attenuates Toll-like receptor signaling via modification of the signaling adaptor TRAF6 and transcription factor NF-κB.

Plexin-B2 and Plexin-D1 in dendritic cells: expression and IL-12/IL-23p40 production.

Characterization of NLRP12 during the in vivo host immune response to Klebsiella pneumoniae and Mycobacterium tuberculosis.

Analysis of the murine immune response to pulmonary delivery of precisely fabricated nano- and microscale particles.

Primary ear fibroblast derivation from mice.

Delayed-type hypersensitivity models in mice.

Bacteria-mediated acute lung inflammation.

Contact hypersensitivity models in mice.

Induction of allergic airway disease using house dust mite allergen.

Cyclic-di-GMP and cyclic-di-AMP activate the NLRP3 inflammasome.

Correction: Characterization of NLRP12 during the In Vivo Host Immune Response to Klebsiella pneumoniae and Mycobacterium tuberculosis.