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Resistance to a bacterial toxin is mediated by removal of a conserved glycosylation pathway required for toxin-host interactions.
The Journal of biological chemistry Nov, 2003 | Pubmed ID: 12944392
Pore worms: using Caenorhabditis elegans to study how bacterial toxins interact with their target host.
International journal of medical microbiology : IJMM Apr, 2004 | Pubmed ID: 15149037
Mitogen-activated protein kinase pathways defend against bacterial pore-forming toxins.
Proceedings of the National Academy of Sciences of the United States of America Jul, 2004 | Pubmed ID: 15256590
Assays for toxicity studies in C. elegans with Bt crystal proteins.
Methods in molecular biology (Clifton, N.J.) , 2006 | Pubmed ID: 16988432
Global functional analyses of cellular responses to pore-forming toxins.
PLoS pathogens Mar, 2011 | Pubmed ID: 21408619
Agilent Technologies
Rachel Formosa1,
Harini Ravi1,
Scott Happe1,
Danielle Huffman1,
Natalia Novoradovskaya1,
Robert Kincaid1,
Steve Garrett1
1, Agilent Technologies
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