Morgan Salmon

Over-expression of the transcription factor, ZBP-89, leads to enhancement of the C2C12 myogenic program.

Cooperative binding of KLF4, pELK-1, and HDAC2 to a G/C repressor element in the SM22α promoter mediates transcriptional silencing during SMC phenotypic switching in vivo.

KLF4 regulates abdominal aortic aneurysm morphology and deletion attenuates aneurysm formation.

Inhibition of interleukin-1β decreases aneurysm formation and progression in a novel model of thoracic aortic aneurysms.

5-Lipoxygenase pathway in experimental abdominal aortic aneurysms.

Attenuation of aortic aneurysms with stem cells from different genders.

Pharmacologic blockade and genetic deletion of androgen receptor attenuates aortic aneurysm formation.

ZFP148 (Zinc-Finger Protein 148) Binds Cooperatively With NF-1 (Neurofibromin 1) to Inhibit Smooth Muscle Marker Gene Expression During Abdominal Aortic Aneurysm Formation.

Klf4, Klf2, and Zfp148 activate autophagy-related genes in smooth muscle cells during aortic aneurysm formation.