Inbal Hazan-Halevy

Stimulation of NADPH oxidase by angiotensin II in human neutrophils is mediated by ERK, p38 MAP-kinase and cytosolic phospholipase A2.

WP1066 disrupts Janus kinase-2 and induces caspase-dependent apoptosis in acute myelogenous leukemia cells.

The effects of natural antioxidants from tomato extract in treated but uncontrolled hypertensive patients.

Kit inhibitor APcK110 induces apoptosis and inhibits proliferation of acute myeloid leukemia cells.

STAT3 is constitutively phosphorylated on serine 727 residues, binds DNA, and activates transcription in CLL cells.

Kit inhibitor APcK110 extends survival in an AML xenograft mouse model.

The synthetic heat shock protein 90 (Hsp90) inhibitor EC141 induces degradation of Bcr-Abl p190 protein and apoptosis of Ph-positive acute lymphoblastic leukemia cells.

STAT-3 activates NF-kappaB in chronic lymphocytic leukemia cells.

Activation of CD44, a receptor for extracellular matrix components, protects chronic lymphocytic leukemia cells from spontaneous and drug induced apoptosis through MCL-1.

The cytokine midkine and its receptor RPTPζ regulate B cell survival in a pathway induced by CD74.

Reciprocal changes in regulatory T cells and Th17 helper cells induced by exercise in patients with chronic lymphocytic leukemia.

Increased CD39 expression on CD4(+) T lymphocytes has clinical and prognostic significance in chronic lymphocytic leukemia.

Divergence in CD19-mediated signaling unfolds intraclonal diversity in chronic lymphocytic leukemia, which correlates with disease progression.

Personalized medicine in CLL: current status and future perspectives.

Stimulation of the B-cell receptor activates the JAK2/STAT3 signaling pathway in chronic lymphocytic leukemia cells.

STAT3-activated GM-CSFRα translocates to the nucleus and protects CLL cells from apoptosis.

Cell-specific uptake of mantle cell lymphoma-derived exosomes by malignant and non-malignant B-lymphocytes.

Metabolism pathways in chronic lymphocytic leukemia.

Harnessing RNAi-based nanomedicines for therapeutic gene silencing in B-cell malignancies.