Andrew D. Chisholm

The C. elegans LAR-like receptor tyrosine phosphatase PTP-3 and the VAB-1 Eph receptor tyrosine kinase have partly redundant functions in morphogenesis.

The divergent C. elegans ephrin EFN-4 functions inembryonic morphogenesis in a pathway independent of the VAB-1 Eph receptor.

The Caenorhabditis elegans vab-10 spectraplakin isoforms protect the epidermis against internal and external forces.

Laminin alpha subunits and their role in C. elegans development.

C. elegans ankyrin repeat protein VAB-19 is a component of epidermal attachment structures and is essential for epidermal morphogenesis.

Intermediate filaments are required for C. elegans epidermal elongation.

Genetic suppression of intronic +1G mutations by compensatory U1 snRNA changes in Caenorhabditis elegans.

The cytoskeleton and epidermal morphogenesis in C. elegans.

Genetic analysis of the Caenorhabditis elegans pax-6 locus: roles of paired domain-containing and nonpaired domain-containing isoforms.

Neurosurgery: functional regeneration after laser axotomy.

Regulation of a DLK-1 and p38 MAP kinase pathway by the ubiquitin ligase RPM-1 is required for presynaptic development.

The two isoforms of the Caenorhabditis elegans leukocyte-common antigen related receptor tyrosine phosphatase PTP-3 function independently in axon guidance and synapse formation.

The C. elegans eyes absent ortholog EYA-1 is required for tissue differentiation and plays partially redundant roles with PAX-6.

Neuronal differentiation in C. elegans.

C. elegans Kallmann syndrome protein KAL-1 interacts with syndecan and glypican to regulate neuronal cell migrations.

The phosphoinositide kinase PIKfyve/Fab1p regulates terminal lysosome maturation in Caenorhabditis elegans.

Gastrulation: Wnts signal constriction.

The PLEXIN PLX-2 and the ephrin EFN-4 have distinct roles in MAB-20/Semaphorin 2A signaling in Caenorhabditis elegans morphogenesis.

Caenorhabditis elegans neuronal regeneration is influenced by life stage, ephrin signaling, and synaptic branching.

Epidermal morphogenesis.

Distinct innate immune responses to infection and wounding in the C. elegans epidermis.

Semaphorin signaling in morphogenesis: found in translation.

The C. elegans F-spondin family protein SPON-1 maintains cell adhesion in neural and non-neural tissues.

The cell signaling adaptor protein EPS-8 is essential for C. elegans epidermal elongation and interacts with the ankyrin repeat protein VAB-19.

Negative regulation of Caenorhabditis elegans epidermal damage responses by death-associated protein kinase.

The DLK-1 kinase promotes mRNA stability and local translation in C. elegans synapses and axon regeneration.

SUMO regulates the assembly and function of a cytoplasmic intermediate filament protein in C. elegans.

Caenorhabditis elegans: a new model organism for studies of axon regeneration.

Calcium and cyclic AMP promote axonal regeneration in Caenorhabditis elegans and require DLK-1 kinase.

Dynamic chromatin organization during foregut development mediated by the organ selector gene PHA-4/FoxA.

The C. elegans peroxidasin PXN-2 is essential for embryonic morphogenesis and inhibits adult axon regeneration.

Axon regeneration mechanisms: insights from C. elegans.

Axon regeneration pathways identified by systematic genetic screening in C. elegans.

A Gαq-Ca²⁺ signaling pathway promotes actin-mediated epidermal wound closure in C. elegans.

Cell identification and cell lineage analysis.

Kinesin-13 and tubulin posttranslational modifications regulate microtubule growth in axon regeneration.

Quantitative semi-automated analysis of morphogenesis with single-cell resolution in complex embryos.

The Caenorhabditis elegans epidermis as a model skin. I: development, patterning, and growth.

The Caenorhabditis elegans epidermis as a model skin. II: differentiation and physiological roles.

Cytoskeletal dynamics in Caenorhabditis elegans axon regeneration.

Mechanisms of ephrin receptor protein kinase-independent signaling in amphid axon guidance in Caenorhabditis elegans.

The EBAX-type Cullin-RING E3 ligase and Hsp90 guard the protein quality of the SAX-3/Robo receptor in developing neurons.

The wounded worm: Using C. elegans to understand the molecular basis of skin wound healing.

Insights into the functions of the death associated protein kinases from C. elegans and other invertebrates.

S6 kinase inhibits intrinsic axon regeneration capacity via AMP kinase in Caenorhabditis elegans.

C. elegans Epidermal Wounding Induces a Mitochondrial ROS Burst that Promotes Wound Repair.

Syndecan defines precise spindle orientation by modulating Wnt signaling in C. elegans.