Edward Mitre

Majority of interferon-gamma-producing CD4+ cells in patients infected with human T cell lymphotrophic virus do not express tax protein.

Lack of basophilia in human parasitic infections.

Parasite antigen-driven basophils are a major source of IL-4 in human filarial infections.

Saturation of immunoglobulin E (IgE) binding sites by polyclonal IgE does not explain the protective effect of helminth infections against atopy.

Basophils, basophilia and helminth infections.

IgE memory: persistence of antigen-specific IgE responses years after treatment of human filarial infections.

Methotrexate as a corticosteroid-sparing agent in complicated neurocysticercosis.

CD4(+) (and not CD25+) T cells are the predominant interleukin-10-producing cells in the circulation of filaria-infected patients.

Microfilariae of the filarial nematode Litomosoides sigmodontis exacerbate the course of lipopolysaccharide-induced sepsis in mice.

Ascaris lumbricoides-induced interleukin-10 is not associated with atopy in schoolchildren in a rural area of the tropics.

Inhibition of type 1 diabetes in filaria-infected non-obese diabetic mice is associated with a T helper type 2 shift and induction of FoxP3+ regulatory T cells.

Evidence for in utero sensitization to Ascaris lumbricoides in newborns of mothers with ascariasis.

Litomosoides sigmodontis: a simple method to infect mice with L3 larvae obtained from the pleural space of recently infected jirds (Meriones unguiculatus).

Type 2 immune-inducing helminth vaccination maintains protective efficacy in the setting of repeated parasite exposures.

Induction of type 2 responses by schistosome worms during prepatent infection.

Basophils amplify type 2 immune responses, but do not serve a protective role, during chronic infection of mice with the filarial nematode Litomosoides sigmodontis.

Immune system development during early childhood in tropical Latin America: evidence for the age-dependent down regulation of the innate immune response.

Impact of early life exposures to geohelminth infections on the development of vaccine immunity, allergic sensitization, and allergic inflammatory diseases in children living in tropical Ecuador: the ECUAVIDA birth cohort study.

Anti-FcεR1 antibody injections activate basophils and mast cells and delay Type 1 diabetes onset in NOD mice.

Helminth protection against autoimmune diabetes in nonobese diabetic mice is independent of a type 2 immune shift and requires TGF-β.

Histamine release and surface CD200R1 staining as sensitive methods for assessing murine mast cell activation.

Chronic helminth infection reduces basophil responsiveness in an IL-10-dependent manner.

Molecular mimicry between cockroach and helminth glutathione S-transferases promotes cross-reactivity and cross-sensitization.

Helminth infection is associated with decreased basophil responsiveness in human beings.

Maternal geohelminth infections are associated with an increased susceptibility to geohelminth infection in children: a case-control study.

Chronic helminth infection does not exacerbate Mycobacterium tuberculosis infection.

Basophils help establish protective immunity induced by irradiated larval vaccination for filariasis.

Vaccination with intestinal tract antigens does not induce protective immunity in a permissive model of filariasis.

A comprehensive, model-based review of vaccine and repeat infection trials for filariasis.

Eosinophilia: a diagnostic clue for nonbacterial diseases in patients with systemic inflammatory response syndrome.

Filaria zoogeography in Africa: ecology, competitive exclusion, and public health relevance.

Worms as therapeutic agents for allergy and asthma: Understanding why benefits in animal studies have not translated into clinical success.

Immunologic characterization of 3 murine regimens of allergen-specific immunotherapy.