Margit Fuchs

Motility enhancement by tumor-derived mutant E-cadherin is sensitive to treatment with epidermal growth factor receptor and phosphatidylinositol 3-kinase inhibitors.

Influence of tumor-associated E-cadherin mutations on tumorigenicity and metastasis.

Effect of wild-type and mutant E-cadherin on cell proliferation and responsiveness to the chemotherapeutic agents cisplatin, etoposide, and 5-fluorouracil.

Dynamics of cell adhesion and motility in living cells is altered by a single amino acid change in E-cadherin fused to enhanced green fluorescent protein.

Effect of tumor-associated mutant E-cadherin variants with defects in exons 8 or 9 on matrix metalloproteinase 3.

Deletion of exon 8 increases cisplatin-induced E-cadherin cleavage.

Enhanced activation of epidermal growth factor receptor caused by tumor-derived E-cadherin mutations.

Rac1 and Rho contribute to the migratory and invasive phenotype associated with somatic E-cadherin mutation.

Identification of the key structural motifs involved in HspB8/HspB6-Bag3 interaction.

Learning cellular texture features in microscopic cancer cell images for automated cell-detection.

A functional interplay between the small GTPase Rab11a and mitochondria-shaping proteins regulates mitochondrial positioning and polarization of the actin cytoskeleton downstream of Src family kinases.

A Role for the Chaperone Complex BAG3-HSPB8 in Actin Dynamics, Spindle Orientation and Proper Chromosome Segregation during Mitosis.