Yanyan Zhang

Effects of aerobic exercise training on large-conductance Ca(2+)-activated K (+) channels in rat cerebral artery smooth muscle cells.

Exercise training reverses unparallel downregulation of MaxiK channel α- and β1-subunit to enhance vascular function in aging mesenteric arteries.

Exercise intensity-dependent reverse and adverse remodeling of voltage-gated Ca(2+) channels in mesenteric arteries from spontaneously hypertensive rats.

Exercise Prevents Upregulation of RyRs-BKCa Coupling in Cerebral Arterial Smooth Muscle Cells From Spontaneously Hypertensive Rats.

Epigenetic regulation of L-type voltage-gated Ca channels in mesenteric arteries of aging hypertensive rats.

Aerobic exercise of low to moderate intensity corrects unequal changes in BK(Ca) subunit expression in the mesenteric arteries of spontaneously hypertensive rats.

BK channel activity and vascular contractility alterations with hypertension and aging via β1 subunit promoter methylation in mesenteric arteries.

Akt modulation by miR-145 during exercise-induced VSMC phenotypic switching in hypertension.

Aerobic exercise enhanced endothelium-dependent vasorelaxation in mesenteric arteries in spontaneously hypertensive rats: the role of melatonin.

Melatonin activates BK channels in cerebral artery myocytes via both direct and MT receptor/PKC-mediated pathway.

Exercise during pregnancy enhances vascular function via epigenetic repression of Ca1.2 channel in offspring of hypertensive rats.

Aerobic exercise-induced inhibition of PKCα/Ca1.2 pathway enhances the vasodilation of mesenteric arteries in hypertension.

Role of the Balance of Akt and MAPK Pathways in the Exercise-Regulated Phenotype Switching in Spontaneously Hypertensive Rats.

Chronic exercise mediates epigenetic suppression of L-type Ca2+ channel and BKCa channel in mesenteric arteries of hypertensive rats.

Prenatal exercise reprograms the development of hypertension progress and improves vascular health in SHR offspring.

Aerobic training-mediated DNA hypermethylation of Agtr1a and Mas1 genes ameliorate mesenteric arterial function in spontaneously hypertensive rats.

Maternal exercise upregulates the DNA methylation of Agtr1a to enhance vascular function in offspring of hypertensive rats.

Maternal exercise represses Nox4 via SIRT1 to prevent vascular oxidative stress and endothelial dysfunction in SHR offspring.

Advances in exercise-induced vascular adaptation: mechanisms, models, and methods.