Isgouhi Kaloshian

Aphid-induced defense responses in Mi-1-mediated compatible and incompatible tomato interactions.

Rme1 is necessary for Mi-1-mediated resistance and acts early in the resistance pathway.

Gene-for-gene disease resistance: bridging insect pest and pathogen defense.

Hemipterans as plant pathogens.

Mi-1-Mediated aphid resistance involves salicylic acid and mitogen-activated protein kinase signaling cascades.

The Mi-9 gene from Solanum arcanum conferring heat-stable resistance to root-knot nematodes is a homolog of Mi-1.

Virus-induced gene silencing in plant roots.

The MI-1-mediated pest resistance requires Hsp90 and Sgt1.

Coil-dependent signaling pathway is not required for Mi-1-mediated potato aphid resistance.

Tomato susceptibility to root-knot nematodes requires an intact jasmonic acid signaling pathway.

Ethylene contributes to potato aphid susceptibility in a compatible tomato host.

Immunity and other defenses in pea aphids, Acyrthosiphon pisum.

WRKY72-type transcription factors contribute to basal immunity in tomato and Arabidopsis as well as gene-for-gene resistance mediated by the tomato R gene Mi-1.

Linked, if not the same, Mi-1 homologues confer resistance to tomato powdery mildew and root-knot nematodes.

The receptor-like kinase SlSERK1 is required for Mi-1-mediated resistance to potato aphids in tomato.

SEED: efficient clustering of next-generation sequences.

SlWRKY70 is required for Mi-1-mediated resistance to aphids and nematodes in tomato.