Judith Hellman

Bacterial peptidoglycan-associated lipoprotein is released into the bloodstream in gram-negative sepsis and causes inflammation and death in mice.

Murein lipoprotein, peptidoglycan-associated lipoprotein, and outer membrane protein A are present in purified rough and smooth lipopolysaccharides.

Protective efficacy of CAP18106-138-immunoglobulin G in sepsis.

Inhaled nitric oxide for ARDS: searching for a more focused use.

Bacterial peptidoglycan-associated lipoprotein: a naturally occurring toll-like receptor 2 agonist that is shed into serum and has synergy with lipopolysaccharide.

Increased leakage of sarcoplasmic reticulum Ca2+ contributes to abnormal myocyte Ca2+ handling and shortening in sepsis.

MyD88-dependent and MyD88-independent pathways in synergy, priming, and tolerance between TLR agonists.

Passive immunization to outer membrane proteins MLP and PAL does not protect mice from sepsis.

Toll-like receptor 2 activation by bacterial peptidoglycan-associated lipoprotein activates cardiomyocyte inflammation and contractile dysfunction.

MyD88-dependent immune activation mediated by human immunodeficiency virus type 1-encoded Toll-like receptor ligands.

Activation of Toll-like receptor 2 impairs hypoxic pulmonary vasoconstriction in mice.

Upregulation of PD-L1 on monocytes and dendritic cells by HIV-1 derived TLR ligands.

Resilience to bacterial infection: difference between species could be due to proteins in serum.

Editorial: Pattern recognition receptors and factor B: "complement"ary pathways converge.

Bacterial lipoprotein TLR2 agonists broadly modulate endothelial function and coagulation pathways in vitro and in vivo.

Activation of endothelial TLR2 by bacterial lipoprotein upregulates proteins specific for the neutrophil response.

ERK5 protein promotes, whereas MEK1 protein differentially regulates, the Toll-like receptor 2 protein-dependent activation of human endothelial cells and monocytes.

Identification of hemopexin as an anti-inflammatory factor that inhibits synergy of hemoglobin with HMGB1 in sterile and infectious inflammation.

Alveolar macrophages and Toll-like receptor 4 mediate ventilated lung ischemia reperfusion injury in mice.

Vascular endothelial cell Toll-like receptor pathways in sepsis.

-Arachidonoyl Dopamine Modulates Acute Systemic Inflammation via Nonhematopoietic TRPV1.

Activation of CBR Promotes Lipopolysaccharide-Induced IL-10 Secretion by Monocytic Myeloid-Derived Suppressive Cells and Reduces Acute Inflammation and Organ Injury.

N-Oleoyl dopamine induces IL-10 via central nervous system TRPV1 and improves endotoxemia and sepsis outcomes.

COVID-19-Associated Lung Microvascular Endotheliopathy: A "From the Bench" Perspective.

Reply to: SARS-CoV2 Endotheliopathy: Insights from Single Cell RNAseq.