Amy E. Birsner

S-3-Amino-phthalimido-glutarimide inhibits angiogenesis and growth of B-cell neoplasias in mice.

Genetic loci that control vascular endothelial growth factor-induced angiogenesis.

Genetic loci that control the angiogenic response to basic fibroblast growth factor.

A 27-amino-acid synthetic peptide corresponding to the NH2-terminal zinc-binding domain of endostatin is responsible for its antitumor activity.

Antiangiogenic effect of oral 2-methoxyestradiol on choroidal neovascularization in mice.

Chronic suppression of angiogenesis following radiation exposure is independent of hematopoietic reconstitution.

Mutant anthrax toxin B moiety (protective antigen) inhibits angiogenesis and tumor growth.

The mouse cornea micropocket angiogenesis assay.

Differential suppression of vascular permeability and corneal angiogenesis by nonsteroidal anti-inflammatory drugs.

Genetic loci that control the size of laser-induced choroidal neovascularization.

Green tea epigallocatechin-3-gallate inhibits angiogenesis and suppresses vascular endothelial growth factor C/vascular endothelial growth factor receptor 2 expression and signaling in experimental endometriosis in vivo.

The classical pink-eyed dilution mutation affects angiogenic responsiveness.

Endostatin lowers blood pressure via nitric oxide and prevents hypertension associated with VEGF inhibition.

Vascular endothelial growth factor C is increased in endometrium and promotes endothelial functions, vascular permeability and angiogenesis and growth of endometriosis.

The albino mutation of tyrosinase alters ocular angiogenic responsiveness.

Melanocyte-secreted fibromodulin promotes an angiogenic microenvironment.